A lipid peroxidative mechanism for posterior subcapsular cataract formation in the rabbit: a possible model for cataract formation in tapetoretinal diseases.
A lipid peroxidative mechanism for posterior subcapsular cataract formation in the rabbit: a possible model for cataract formation in tapetoretinal diseases.
复制标题
兔子后囊下白内障形成的脂质过氧化机制：绦虫视网膜疾病中白内障形成的可能模型。
DOI：
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发表时间：
1984
影响因子：
4.4
通讯作者：
C. Garcia
中科院分区：
文献类型：
--
作者：
J. Goosey;W. Tuan;C. Garcia
注射二十二碳六烯酸后，兔子出现后囊下白内障。通过丙二醛的形成和玻璃体荧光证明了二十二碳六烯酸在兔玻璃体中的自氧化。后囊下白内障形成的发生在时间上与玻璃体丙二醛水平下降相关。
Posterior subcapsular cataracts developed in rabbits following injection of docosahexenoic acid. Autoxidation of docosahexenoic acid in rabbit vitreous was demonstrated by the formation of malondialdehyde and vitreous fluorescence. The onset of posterior subcapsular cataract formation was temporally correlated with declining levels of vitreous malondialdehyde.