Reduced immobilizing properties of isoflurane and nitrous oxide in mutant mice lacking theN-methyl-D-aspartate receptor GluRepsilon1 subunit are caused by the secondary effects of gene knockout.
Reduced immobilizing properties of isoflurane and nitrous oxide in mutant mice lacking theN-methyl-D-aspartate receptor GluRepsilon1 subunit are caused by the secondary effects of gene knockout.
复制标题
缺乏N-甲基-D-天冬氨酸受体GluRepsilon1亚基的突变小鼠中异氟烷和一氧化二氮的固定特性降低是由基因敲除的二次效应引起的。
DOI：
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发表时间：
2010
期刊：
影响因子：
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通讯作者：
et al.
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作者：
Petrenko AB;et al.
