[A case of basilar artery fenestration with recurrent attacks of vertebrobasilar insufficiency].

[A case of basilar artery fenestration with recurrent attacks of vertebrobasilar insufficiency].
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基底动脉开窗并发椎基底动脉供血不足反复发作一例[J].

DOI:
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发表时间:
1987
期刊:
No to shinkei = Brain and nerve
影响因子:
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通讯作者:
T. Shima
T. Shima
中科院分区:
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文献类型:
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作者:
S. Uchiyama;T. Yoshinaga;T. Shima

文献摘要

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基底动脉穿孔是一种少见的血管异常,通常是尸检或血管造影的偶然产物。除了蛛网膜下腔出血伴囊状动脉瘤外,文献中没有一例出现与此异常相关的临床症状。我们报告一例罕见的基底动脉开窗与临床症状没有任何动脉瘤。患者男,71岁,因反复发作眩晕、恶心、复视、步态不稳等症状,5年来一直接受不稳定性高血压治疗。1985年13号入院前一天,他突然感到复视和眩晕,步态不稳。他的家人注意到他有构音障碍。入院时,他警觉且血压正常。他主诉口周区右半部和右手手指感觉迟钝。神经系统检查显示,他的右腿有轻度虚弱和深腱反射过度活跃。他的运动协调几乎正常,但当他闭着眼睛单脚站立时,他是不稳定的。除血清尿酸水平升高外,实验室检查均正常。胸部X线片显示主动脉硬化改变和轻度心脏扩大。在他的ECG上观察到左心室肥大。他的CT扫描显示多个腔隙和轻度脑萎缩。在脑血管造影中,他的基底动脉(BA)几乎在其全长上有一个开窗,将BA分成两个直径比正常小的部分,就像一个复制品。(250字处删节)
Fenestration of basilar artery is an uncommon vascular anomaly that is usually an incidental product on autopsy or angiography. None of the cases in the literature had clinical symptoms associated with this anomaly except for subarachnoid hemorrhage when accompanied with saccular aneurysm. We report a rare case of the basilar artery fenestration associated with clinical symptoms without any aneurysm. A 71-years-old male, who had been treated for labile hypertension and had had recurrent attacks of vertigo, nausea, sometimes diplopia or unsteady gait, for 5 years, was referred to our hospital on Sept. 13, 1985. One day prior to admission, he suddenly felt diplopia and vertigo and unsteady gait. His family noticed he was dysarthric. On admission, he was alert and normotensive. He complained of dysesthesia on the right half of the perioral region and his right fingers. A neurological examination showed a mild weakness and hyperactive deep tendon reflexes on his right leg. His motor coordination was almost normal, but he was unsteady when he stood on one foot with his eyes closed. Laboratory examinations were normal except for an elevated serum uric acid level. A chest x-ray film showed a sclerotic change of aorta and mild cardiomegaly. Left ventricular hypertrophy was observed on his ECG. His CT scans showed multiple lacunae and mild brain atrophy. On cerebral angiography, his basilar artery (BA) had a fenestration almost in its total length that divided the BA, like a duplication, into two components with a smaller diameter than normal.(ABSTRACT TRUNCATED AT 250 WORDS)