Salicylic acid potentiates an agonist-dependent gain control that amplifies pathogen signals in the activation of defense mechanisms
Salicylic acid potentiates an agonist-dependent gain control that amplifies pathogen signals in the activation of defense mechanisms
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DOI：
10.1105/tpc.9.2.261
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发表时间：
1997-02-01
期刊：
影响因子：
11.6
通讯作者：
Lamb, C
中科院分区：
文献类型：
--
作者：
Shirasu, K;Nakajima, H;Lamb, C
苯丙烷衍生的天然产物水杨酸（SA）在抗病中起着关键作用。然而，SA管理的病原体的情况下是一个矛盾的弱诱导信号，往往需要0.5至5 mM的浓度，以诱导获得性抗性或相关的防御机制或预处理信号系统。相比之下，内源性SA累积至浓度为
The phenylpropanoid-derived natural product salicylic acid (SA) plays a key role in disease resistance. However, SA administered in the absence of a pathogen is a paradoxically weak inductive signal, often requiring concentrations of 0.5 to 5 mM to induce acquired resistance or related defense mechanisms or to precondition signal systems. In contrast, endogenous SA accumulates to concentrations of