A redefinition of the syndrome of Broca's aphasia: Implications for a neuropsychological model of language
A redefinition of the syndrome of Broca's aphasia: Implications for a neuropsychological model of language
复制标题
布罗卡失语症的重新定义：对语言神经心理学模型的启示
DOI：
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发表时间：
1980
影响因子：
2.1
通讯作者：
A. Caramazza
中科院分区：
文献类型：
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作者：
R. Berndt;A. Caramazza
摘要提出了一种神经心理学理论来解释Broca失语综合征。对文献的批判性回顾，着重于最近的研究，为重新定义综合征提供了基础，大大扩大了其经典描述。提出神经心理学解释的重点应该是理论上可分离的心理机制，在局灶性脑损伤的情况下，这些心理机制可能在与其他成分相对隔离的情况下被破坏，而不是在失语表现的孤立单位上。Broca失语症的症状被解释为可预见的行为表现，即语言系统的句法分析成分的中央中断，加上(理论上独立的)发音障碍，仅影响语音输出系统。这一论点的神经解剖学含义是在经典的“强烈地方主义”假说的框架内考虑的。
ABSTRACT A neuropsychological theory is offered to account for the syndrome of Broca's aphasia. A critical review of the literature, with emphasis on recent research, provides the basis for a redefinition of the syndrome that considerably broadens its classical description. The argument is advanced that the focus of neuropsychological explanation should be on theoretically separable psychological mechanisms that might be disrupted in relative isolation from other components in conditions of focal brain damage, rather than on isolated units of aphasic performance. The symptoms that characterize Broca's aphasia are explained as predictable behavioral manifestations of a central disruption of the syntactic parsing component of the language System, coupled with a (theoretically independent) articulatory deficit that affects only the speech output System. The neuroanatomical implications of this argument are considered within the framework of the classical “strong localizationist” hypothesis.