Novel splicing variant of mouse Orc1 is deficient in nuclear transolocation and resistant for proteasome-mediated degradation

Novel splicing variant of mouse Orc1 is deficient in nuclear transolocation and resistant for proteasome-mediated degradation
复制标题

小鼠 Orc1 的新型剪接变体缺乏核易位并且对蛋白酶体介导的降解具有抵抗力

DOI:
--
复制
发表时间:
2005
期刊:
The Journal of Biological Chmeistry 280・13
影响因子:
--
通讯作者:
F.Hanaoka
F.Hanaoka
中科院分区:
--
文献类型:
--
作者:
Y.Miyake;T.Mizuno;K.Yanagi;F.Hanaoka

文献摘要

相似文献