Novel splicing variant of mouse Orc1 is deficient in nuclear transolocation and resistant for proteasome-mediated degradation
Novel splicing variant of mouse Orc1 is deficient in nuclear transolocation and resistant for proteasome-mediated degradation
复制标题
小鼠 Orc1 的新型剪接变体缺乏核易位并且对蛋白酶体介导的降解具有抵抗力
DOI:
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发表时间:
2005
期刊:
影响因子:
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通讯作者:
F.Hanaoka
中科院分区:
文献类型:
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作者:
Y.Miyake;T.Mizuno;K.Yanagi;F.Hanaoka