Diversity in valuing social contact and risk tolerance leading to the emergence of homophily in populations facing infectious threats
Diversity in valuing social contact and risk tolerance leading to the emergence of homophily in populations facing infectious threats
复制标题
重视社会接触和风险承受能力的多样性导致面临感染威胁的人群出现同质性
DOI：
10.1103/physreve.105.044315
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发表时间：
2022
影响因子：
2.4
通讯作者：
Fefferman, Nina H.
中科院分区：
文献类型：
--
作者：
Young, Matthew J.;Silk, Matthew J.;Pritchard, Alex J.;Fefferman, Nina H.
自组织如何导致社会群体结构的出现仍然是复杂系统研究中一个令人着迷且开放的问题。跨系统反复出现的一种经常观察到的结构是自相似群落，即同质性。我们使用博弈论的视角来探讨个人仅基于两个因素选择隶属伙伴关系的情况：他们对社交接触的重视程度，以及他们对社交接触带来的威胁（例如传染病、威胁性想法等）的风险承受能力。我们展示了这两种影响的多样性如何足以导致群体中自组织同质性的出现。我们进一步考虑外部社会因素影响维持特定社会关系的愿望的情况，并显示新兴的同质性模式对这些额外影响的稳健性。这些结果表明，在平衡社会接触的价值和与这些接触相关的潜在风险的个人行为中，可能会出现可观察到的人群水平的同质性。我们在人群中爆发传染病的背景下展示并讨论这些结果。作为对社会分工如何改变流行病学风险的标准叙述的补充，我们在这里展示了流行病学风险如何加深人群中的社会分工。
How self-organization leads to the emergence of structure in social populations remains a fascinating and open question in the study of complex systems. One frequently observed structure that emerges again and again across systems is that of self-similar community, i.e., homophily. We use a game theoretic perspective to explore a case in which individuals choose affiliation partnerships based on only two factors: the value they place on having social contacts, and their risk tolerance for exposure to threat derived from social contact (e.g., infectious disease, threatening ideas, etc.). We show how diversity along just these two influences is sufficient to cause the emergence of self-organizing homophily in the population. We further consider a case in which extrinsic social factors influence the desire to maintain particular social ties, and show the robustness of emergent homophilic patterns to these additional influences. These results demonstrate how observable population-level homophily may arise out of individual behaviors that balance the value of social contacts against the potential risks associated with those contacts. We present and discuss these results in the context of outbreaks of infectious disease in human populations. Complementing the standard narrative about how social division alters epidemiological risk, we here show how epidemiological risk may deepen social divisions in human populations.