Transgenic overexpression of G5PR that is normally augmented in centrocytes impairsthe enrichment of high-affinity antigen-specific B cells, increases peritoneal B-1a cells, and induces autoimmunity in aged female mice
Transgenic overexpression of G5PR that is normally augmented in centrocytes impairsthe enrichment of high-affinity antigen-specific B cells, increases peritoneal B-1a cells, and induces autoimmunity in aged female mice
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通常在中心细胞中增强的 G5PR 转基因过表达会损害高亲和力抗原特异性 B 细胞的富集，增加腹膜 B-1a 细胞，并诱导老年雌性小鼠的自身免疫
DOI：
10.4049/jimmunol.1102774
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发表时间：
2012
期刊：
影响因子：
--
通讯作者：
and Sakaguchi N
中科院分区：
文献类型：
--
作者：
hi H;Ohtsuji M;Tsurui H;Hirose S;and Sakaguchi N
