Loss of the inhibitory function of the guanine nucleotide regulatory component of adenylate cyclase due to its ADP ribosylation by islet-activating protein, pertussis toxin, in adipocyte membranes.
Loss of the inhibitory function of the guanine nucleotide regulatory component of adenylate cyclase due to its ADP ribosylation by islet-activating protein, pertussis toxin, in adipocyte membranes.
复制标题
由于脂肪细胞膜中的胰岛激活蛋白（百日咳毒素）对腺苷酸环化酶的 ADP 核糖基化，导致腺苷酸环化酶的鸟嘌呤核苷酸调节成分的抑制功能丧失。
DOI：
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复制
发表时间：
1983
影响因子：
4.8
通讯作者：
M. Ui
中科院分区：
文献类型：
--
作者：
T. Murayama;M. Ui
