Androgen receptor null male mice develop late-onset obesity due to decreased energy expenditure and lypolytic activity but show normal insulin sensitivity with high adiponectin secretion.
Androgen receptor null male mice develop late-onset obesity due to decreased energy expenditure and lypolytic activity but show normal insulin sensitivity with high adiponectin secretion.
复制标题
雄激素受体缺失的雄性小鼠由于能量消耗和脂肪分解活性降低而出现迟发性肥胖,但显示出正常的胰岛素敏感性和高脂联素分泌。
DOI:
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发表时间:
2005
期刊:
影响因子:
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通讯作者:
Nawata H
中科院分区:
文献类型:
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作者:
Fan W;Yanase T;Nomura M;Okabe T;Goto K;Sato T;Kawano H;Kato S;Nawata H