Posttranslational modification of mammalian AP endonuclease (APE1).
Posttranslational modification of mammalian AP endonuclease (APE1).
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DOI:
10.1007/s00018-010-0487-3
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发表时间:
2010-11
影响因子:
8
通讯作者:
Izumi, Tadahide
中科院分区:
文献类型:
--
作者:
Busso, Carlos S.;Lake, Michael W.;Izumi, Tadahide
A key issue in studying mammalian DNA base excision repair is how its component proteins respond to a plethora of cell-signaling mediators invoked by DNA damage and stress-inducing agents such as reactive oxygen species, and how the actions of individual BER proteins are attributed to cell survival or apoptotic/necrotic death. This article reviews the past and recent progress on posttranslational modification (PTM) of mammalian apurinic/apyrimidinic (AP) endonuclease 1 (APE1).
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影响因子:
11.5
作者:
Bobola, MS;Finn, LS;Silber, JR
通讯作者:
Silber, JR
DOI:
10.1016/j.mrfmmm.2008.04.008
发表时间:
2008-08-25
影响因子:
2.3
作者:
Georgiadis, M. M.;Luo, M.;Kelley, M. R.
通讯作者:
Kelley, M. R.
DOI:
10.1073/pnas.88.24.11450
发表时间:
1991-12-01
影响因子:
11.1
作者:
DEMPLE, B;HERMAN, T;CHEN, DS
通讯作者:
CHEN, DS
影响因子:
64.8
作者:
Dornan, D;Wertz, I;Dixit, VM
通讯作者:
Dixit, VM
影响因子:
3.8
作者:
Daviet, Stephane;Couve-Privat, Sophie;Ishchenko, Alexander A.
通讯作者:
Ishchenko, Alexander A.