Dental pulp exposed to the CO2 laser beam

Dental pulp exposed to the CO2 laser beam
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牙髓暴露于 CO2 激光束

DOI:
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发表时间:
1987
影响因子:
2.4
通讯作者:
Dds F. Melcer
Dds F. Melcer
中科院分区:
医学3区
文献类型:
--
作者:
Dds J. Melcer;MD M.;Dds F. Melcer

文献摘要

被引文献

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通过显微镜和显微射线照相术,在 5 天、1 个月和 3 个月后研究了比格犬和猕猴的牙齿(牙本质和牙髓)上应用 CO2 激光束引起的创伤性炎症,刺激了牙本质牙髓组织的形态学现象。牙本质暴露于 2 × 103 J/cm2 的能量密度后,牙髓组织的第一层细胞显示出稀疏和细胞变性,随后由于成牙本质细胞的激发或之前功能的牙髓细胞产生,在 3 个月内形成约 300 μm 厚的钙化牙本质。将 103J/cm2 数量级的能量密度应用于牙髓珠,使其部分坏死、各种炎症,并在 1 个月时通过 200 μm 的新牙本质桥实现准恒定再生。细胞活性的分析似乎可以通过这种方法进行研究。
Traumatic inflammation due to application of CO2 laser beam on teeth (dentin and pulp) of Beagle dogs and Macaca Monkeys, stimulate in dentino‐pulpal tissue morphological phenomena studied after 5 days, 1, and 3 months by microscopy and microradiography. After a dentin exposure to a density of energy from 2 × 103 J/cm2, the first cell layers of the pulp tissue show a rarefaction and a cellular degeneration, followed by a neoformation of cacified dentin, of about 300 μm thick in 3 months, due to the excitation of odontoblasts or produced by pulpal cells functioning before. A density of energy of 103J/cm2 order applied to the pulp beads to its partial necrosis, to various inflammatory aspects and to a quasi‐constant regeneration by a neo‐dentin bridge of 200 μm at 1 month. The analysis of cell activity seems able to be investigated from this method.