Amygdaloid modulation of sodium chloride and water intake in the rat.
Amygdaloid modulation of sodium chloride and water intake in the rat.
复制标题
杏仁核对大鼠氯化钠和水摄入的调节。
DOI：
10.1037/h0031377
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发表时间：
1971
期刊：
影响因子：
--
通讯作者：
E. Chiaraviglio
中科院分区：
文献类型：
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作者：
E. Chiaraviglio
对成年雄性白化大鼠进行5次实验。单侧乙酰胆碱作用于无钠大鼠的杏仁核，增加了大鼠的nacl摄入量，而减少了口渴和饱足大鼠的nacl摄入量。双侧横断终纹（st）或腹侧杏仁核束（vaf）可使无钠大鼠的生理盐水摄入量减少。在无钠大鼠中，横断终纹或腹侧杏仁核束可使钠摄入量减少。有st病变的ss水分摄取量增加。无钠大鼠的杏仁核中乙酰胆碱含量升高，钠摄入量达到控制水平。横断两条通路加上杏仁核刺激不能增加生理盐水的摄入量。由此得出结论，杏仁核的脉冲通过st和vaf到达下丘脑调节盐的摄入量。（PsycINFO数据库记录(c) 2016 APA，版权所有）
Conducted 5 experiments with adult male albino rats. Unilateral application of acetylcholine to the amygdala in sodium-deprived ss increased nacl intake while decreasing it in thirsty and satiated ss. Water intake was not affected. Bilateral transection of stria terminalis (st) or ventral amygdalofugal (vaf) tract, decreased saline intake in sodium-deprived ss. In sated ss, damage to st did not affect sodium intake, while vaf-tract transection decreased it. Water intake increased in sated ss with st lesions. Acetylcholine in the amygdala of sodium-deprived ss with damage to either tract raised sodium intake to control levels. Transection of both pathways plus amygdaloid stimulation failed to increase saline intake. It is concluded that impulses from amygdala reaching hypothalamus through st and vaf modulate nacl intake.(PsycINFO Database Record (c) 2016 APA, all rights reserved)