Muscular dystrophy with laminin deficiency decreases the content f butyrylcholinesterase tetramers in sciatic nerves of Lama2dy mice
Muscular dystrophy with laminin deficiency decreases the content f butyrylcholinesterase tetramers in sciatic nerves of Lama2dy mice
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DOI:
10.1016/s0304-3940(02)00885-6
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发表时间:
2002-10-18
影响因子:
2.5
通讯作者:
Campoy, FJ
中科院分区:
文献类型:
--
作者:
Moral-Naranjo, MT;Cabezas-Herrera, J;Campoy, FJ
The Lama2dy mouse, a model of congenital muscular dystrophy (DMD) by merosin deficiency (MCMD), shows muscle degeneration and dysmyelination of peripheral nerves. Although it has been reported that MCMD reduces acetylcholinesterase (AChE) activity of mouse sciatic nerve, no information is available regarding its action on butyrylcholinesterase (BuChE). Amphiphilic BuChE monomers (G(1)(A), 39%), dimers (G(2)(A), 18%), and tetramers (G(4)(A), 33%), along with hydrophilic tetramers (G(4)(H), 10%), were identified in mouse sciatic nerve. It also contained abundant G(4)(A) (75%) and less G(1)(A), G(2)(A), G(4)(H) and A(12) AChE components. In dystrophic nerves, the BuChE activity increased 2-fold but the proportion of the G(4)(A) form dropped from 33% to 10%. AChE activity decreased and the composition of enzyme forms was unaffected. Lectin interaction studies showed that, in contrast to skeletal muscle, the defect of merosin did not greatly alter the glycosylation of nerve cholinesterases. The anomalous synthesis of BuChE forms in dystrophic nerve may be related with peripheral neuropathy of MCMD. (C) 2002 Published by Elsevier Science Ireland Ltd.