Brains that are out of tune but in time
Brains that are out of tune but in time
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DOI：
10.1111/j.0956-7976.2004.00683.x
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发表时间：
2004-05-01
影响因子：
8.2
通讯作者：
Peretz, I
中科院分区：
文献类型：
--
作者：
Hyde, KL;Peretz, I
据估计，大约4%的一般人口可能有失音症（或音调聋）。先天性失乐症是一种终身残疾，尽管正常的智力，记忆力和语言能力处理音乐。在这里，我们提出的证据表明，这种疾病源于在细粒度的音高感知赤字。将五个音调的单调和等频序列呈现给音乐和对照成年人（即，固定音高和音程）。他们被要求检测第四个音在音高或时间上的位移。所有的音乐失乐症患者在检测音高变化方面都受到了损害，并且没有表现出通过练习而改善的迹象。相比之下，他们检测到的时间变化以及控制成年人，并表现出类似的改善与实践。因此，在失音乐症个体中看到的音高感知退化不能归因于任务的非特异性问题或一般的听力差。相反，数据表明存在先天性神经异常，选择性地损害音高处理。
It is estimated that about 4% of the general population may have amusia (or tone deafness). Congenital amusia is a lifelong disability for processing music despite normal intellectual, memory, and language skills. Here we present evidence that the disorder stems from a deficit in fine-grained pitch perception. Amusic and control adults were presented with monotonic and isochronous sequences of five tones (i.e., constant pitch and intertone interval). They were required to detect when the fourth tone was displaced in pitch or time. All amusic participants were impaired in detecting the pitch changes, and showed no sign of improvement with practice. In contrast, they detected time changes as well as control adults and exhibited similar improvements with practice. Thus, the degraded pitch perception seen in the amusic individuals cannot be ascribed to nonspecific problems with the task or to poor hearing in general. Rather, the data point to the presence of a congenital neural anomaly that selectively impairs pitch processing.