Multiple mechanisms of extraocular muscle "overaction"
Multiple mechanisms of extraocular muscle "overaction"
复制标题
DOI：
10.1001/archopht.124.5.680
复制
发表时间：
2006-05-01
影响因子：
--
通讯作者：
Kushner, BJ
中科院分区：
文献类型：
--
作者：
Kushner, BJ
目的：分配更具体的病理生理过程的千变万化的模式的眼外肌“过度反应”，我们看到在临床practice.Methods：通过外推从已知的横纹肌生理学的原则，一个有凝聚力的理论关于眼外肌behaviors.Results：理解明显的眼外肌过度反应的关键是要区分的肌肉，弹性下降，一个是加强。原发性下斜肌功能亢进具有肌肉弹性降低的特征，上级直肌功能亢进/收缩综合征似乎代表肌肉增强，继发于同侧上级斜肌麻痹的下斜肌功能亢进具有弹性降低和增强的因素。许多运动模式，似乎是由于过度的肌肉，实际上可能是由其他肌肉比可疑的one.Conclusion：明显的眼外肌过度反应，可以引起许多不同的因素。
Objective: To assign more specific pathophysiologic processes to the protean patterns of extraocular muscle "overaction" that we see in clinical practice.Methods: By extrapolating from known principles of striated muscle physiology, a cohesive theory about extraocular muscle behavior is derived.Results: The key to understanding apparent extraocular muscle overaction is to differentiate between a muscle that has decreased elasticity and one that is strengthened. Primary inferior oblique muscle overaction has the characteristics of a muscle that primarily has decreased elasticity, the superior rectus overaction/contraction syndrome appears to represent a muscle that is strengthened, and inferior oblique overaction secondary to ipsilateral superior oblique palsy has elements of both decreased elasticity and strengthening. Many motility patterns that appear to be due to an overacting muscle may in fact be caused by other muscles than the suspected one.Conclusion: Apparent extraocular muscle overaction can be caused by many different factors.