NMI mediates transcription-independent ARF regulation in response to cellular stresses.
NMI mediates transcription-independent ARF regulation in response to cellular stresses.
复制标题
NMI 介导转录独立的 ARF 调节以应对细胞应激
DOI：
10.1091/mbc.e12-04-0304
复制
发表时间：
2012-12
影响因子：
3.3
通讯作者：
Zhang SQ
中科院分区：
文献类型：
--
作者：
Li Z;Hou J;Sun L;Wen T;Wang L;Zhao X;Xie Q;Zhang SQ
ETOC：Nmi是在酵母双杂交筛选中发现的一种新的ARF相互作用蛋白。NMI可抑制Ulf诱导的ARF蛋白泛素降解。它介导非转录依赖的ARF调节，是稳定和上调ARF对细胞压力的反应所必需的。
ETOC: NMI is a novel ARF-interacting protein identified in a yeast two-hybrid screen. NMI inhibits ULF-induced ubiquitin degradation of ARF protein. It mediates transcription-independent ARF regulation and is required for the stabilization and up-regulation of ARF in response to cellular stresses.