HEART-DISEASE MORTALITY FOLLOWING WIDOWHOOD - SOME RESULTS FROM THE OPCS LONGITUDINAL-STUDY
HEART-DISEASE MORTALITY FOLLOWING WIDOWHOOD - SOME RESULTS FROM THE OPCS LONGITUDINAL-STUDY
复制标题
DOI：
10.1016/0022-3999(87)90052-3
复制
发表时间：
1987-01-01
影响因子：
4.7
通讯作者：
JONES, DR
中科院分区：
文献类型：
--
作者：
JONES, DR
许多研究表明，经历“压力”生活事件后，心肌梗塞、事故和其他疾病的风险可能会升高。在OPCS纵向研究中，定期收集死亡数据，并将1971-1981年期间英格兰和威尔士人口中1%的配偶死亡样本与1971年样本成员的人口普查记录联系在一起。因此，可以分析配偶丧亲这一可能非常紧张的事件之后的死亡时间和模式。总的来说，死亡率（缺血性心脏病）比LS样本中所有成员的死亡率高出不到10%。正如许多早期的研究一样，观察到丧偶后不久死亡率有所上升。不同寻常的是，就各种原因造成的死亡而言，寡妇的这种增加比鳏夫更为明显，例如，在守寡后的第一个月，各种原因造成的死亡率增加了两倍，例如，在守寡后的第一个月，各种原因造成的死亡率增加了两倍。然而，在男性和女性中都没有明确确定死于缺血性心脏病的死亡高峰。尽管这项研究规模很大，而且选择了一个精心挑选的对照组，但从人口普查中收集的数据中，只有有限的研究成员特征是可能的。特别是，没有人格、行为或饮食的衡量标准。对社会或家庭支持的潜在影响的调查，以家庭结构和子女数量来衡量，得出了模棱两可的结果。研究了死亡率上升的几种可能的解释。基于共同婚姻环境、同性婚姻或同时意外死亡的假设被认为价值非常有限。观察到的模式，虽然与压力生活事件的早期影响一致，但并不表明丧亲之痛后的压力会导致缺血性心脏病死亡率过高。
Many studies have suggested that following the experience of ''stressful'' life events the risks of myocardial infarction, accidents and perhaps other diseases are elevated. In the OPCS Longitudinal Study routinely collected data on deaths, and deaths of a spouse occurring in a 1% sample of the population of England and Wales in the period 1971-1981 are linked together, and with 1971 census records of sample members. The timing and patterns of death following the potentially very stressful event of conjugal bereavement may thus be analysed. Overall the mortality (from ischaemic heart disease) was less than 10% in excess of that in all members of the LS sample. As in many earlier studies, some increases in death rates shortly after widowhood are observed. Unusually, for deaths from all causes these increases are more marked in widows than in widowers with, for example, a two-fold increase in mortality from all causes in the first month after in widowers with, for example, a two-fold increase in mortality from all causes in the first month after widowhood. However, no peak of post-bereavement mortality from ischaemic heart disease is clearly established in either sex. Although the study is large, with a well-chosen control group, only a limited characterisation of study members from data collected in the census is possible. In particular, no measures of personality, behaviour or diet are available. Investigation of potential effects of social or familial support, as measured by household structure and numbers of children, led to equivocal results. Several possible explanations for the increased mortality rates are examined. Hypotheses based on common marital environment, homogamy or simultaneous accidental death are seen to be of very limited value. The observed patterns, although consistent with an early effect of a stressful life event, do not suggest that stress following bereavement leads to an excess of ischaemic heart disease mortality.