Inactivation of pertussis toxin-sensitive guanyl nucleotide-binding proteins increase parathyroid hormone receptors and reverse agonist-induced receptor down-regulation in ROS 17/2.8 cells.
Inactivation of pertussis toxin-sensitive guanyl nucleotide-binding proteins increase parathyroid hormone receptors and reverse agonist-induced receptor down-regulation in ROS 17/2.8 cells.
复制标题
百日咳毒素敏感鸟苷酸结合蛋白的失活会增加甲状旁腺激素受体并逆转 ROS 17/2.8 细胞中激动剂诱导的受体下调。
DOI：
10.1210/endo-125-5-2594
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发表时间：
1989
期刊：
影响因子：
4.8
通讯作者：
Segre,GV
中科院分区：
文献类型：
--
作者：
Abou-Samra,AB;Jueppner,H;PottsJr,JT;Segre,GV
