Sleep apnea, hypersomnolence, and upper airway obstruction secondary to adenotonsillar enlargement.
Sleep apnea, hypersomnolence, and upper airway obstruction secondary to adenotonsillar enlargement.
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睡眠呼吸暂停、嗜睡和继发于腺样体扁桃体增大的上呼吸道阻塞。
DOI：
10.1001/archotol.1977.00780240041003
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发表时间：
1977
影响因子：
--
通讯作者：
R. Smith
中科院分区：
文献类型：
--
作者：
D. Mangat;W. Orr;R. Smith
作者检查了4名儿童，他们的扁桃体腺样体肥大导致了间歇性的严重上气道阻塞发作，并通过整夜睡眠和呼吸的多导睡眠图监测进行了记录。先前未报告的严重腺样体扁桃体肥大的并发症包括长时间阻塞性呼吸暂停发作和睡眠模式紊乱。孩子们也表现出白天嗜睡。多导睡眠研究后腺样体切除术证实改善睡眠模式和缓解阻塞发作。我们的患者没有发生肺动脉高压、肺心病或其他报告的长期上呼吸道阻塞并发症，所有这些都可以通过早期诊断和治疗来避免。
The authors examined four children with substantial adenotonsillar enlargement that caused intermittent severe upper airway obstructive episodes, documented by all-night polygraphic monitoring of sleep and respirations. Previously unreported complications of severe adenotonsillar enlargement that were demonstrated included prolonged obstructive apneic episodes and disturbed sleep patterns. The children also exhibited daytime hypersomnolence. Polygraphic sleep studies after adenotonsillectomy confirmed improved sleep patterns and relief of the obstructive episodes. None of our patienqs had developed pulmonary hypertension, cor pulmonale, or other reported complications of prolonged upper airway obstruction, all of which may have been averted by early diagnosis and treatment.