sll1961 is a novel regulator of phycobilisome degradation during nitrogen starvation in the cyanobacterium Synechocystis sp PCC 6803
sll1961 is a novel regulator of phycobilisome degradation during nitrogen starvation in the cyanobacterium Synechocystis sp PCC 6803
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DOI：
10.1016/j.febslet.2008.02.063
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发表时间：
2008-04-02
期刊：
影响因子：
3.5
通讯作者：
Sonoike, Kintake
中科院分区：
文献类型：
--
作者：
Sato, Hanayo;Fujimori, Tamaki;Sonoike, Kintake
sll 1961基因被报道编码蓝细菌集胞藻PCC 6803中的光系统化学计量的调节因子。我们在这里表明，sll1961基因也是必不可少的氮饥饿期间的藻胆体降解。在sll 1961突变体中观察到藻胆体降解的缺陷，尽管nhlA（一种在氮饥饿期间参与藻胆体降解的基因）的表达增加。光系统化学计量不受氮饥饿的sll1961突变体，也不是在野生型。结果表明，存在一种新的途径藻胆体降解控制独立的nblA表达。(C)2008年欧洲生物化学学会联合会。Elsevier B.V.出版，保留所有权利。
The sll1961 gene was reported to encode a regulatory factor of photosystem stoichiometry in the cyanobacterium Synechocystis sp. PCC 6803. We here show that the sll1961 gene is also essential for the phycobilisome degradation during nitrogen starvation. The defect in phycobilisome degradation was observed in the sll1961 mutant despite the increased expression of nhlA, a gene involved in phycobilisome degradation during nitrogen starvation. Photosystem stoichiometry is not affected by nitrogen starvation in the sll1961 mutant nor in the wild-type. The results indicate the presence of a novel pathway for phycobilisome degradation control independent of nblA expression. (C) 2008 Federation of European Biochemical Societies. Published by Elsevier B.V. All rights reserved.