The enhancement of Stanniocalcin-1 (STC1) secretion increases the ability of mesenchymal stem cells (MSCs) to reduce bleomycin-induced lung fibrosis in a mouse model through inhibition of endoplasmic reticulum stress (ER-stress)/TGFbeta1 pathway
The enhancement of Stanniocalcin-1 (STC1) secretion increases the ability of mesenchymal stem cells (MSCs) to reduce bleomycin-induced lung fibrosis in a mouse model through inhibition of endoplasmic reticulum stress (ER-stress)/TGFbeta1 pathway
复制标题
在小鼠模型中,斯钙素-1 (STC1) 分泌的增强可增加间充质干细胞 (MSC) 通过抑制内质网应激 (ER-stress)/TGFbeta1 途径减少博莱霉素诱导的肺纤维化的能力
DOI:
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发表时间:
2014
期刊:
影响因子:
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通讯作者:
Manabu Ono and Masakazu Ichinose
中科院分区:
文献类型:
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作者:
Shinya Ohkouchi;Manabu Ono and Masakazu Ichinose