Brain gangliosides in drug‐induced status epilepticus

Brain gangliosides in drug‐induced status epilepticus
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药物引起的癫痫持续状态中的脑神经节苷脂

DOI:
10.1111/j.1471-4159.1975.tb07681.x
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发表时间:
1975
影响因子:
4.7
通讯作者:
H. Bernheimer
H. Bernheimer
中科院分区:
医学2区
文献类型:
--
作者:
E. Winter;H. Bernheimer

文献摘要

被引文献

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WHISLER et al.(1968)报告称,戊四唑或美解眠诱导犬癫痫持续状态时,脑神经节苷脂浓度降低。在他们的研究中,没有给出在这些实验条件下单个神经节苷脂行为的数据。我们研究了药物诱导的癫痫持续状态对另一种动物兔脑中神经节苷脂浓度和神经节苷脂模式的影响,并简要介绍我们的结果。
WHISLER et al. (1968) have reported that in the dog there is a decrease in the brain ganglioside concentration in status epilepticus induced by pentylenetetrazol or bemegride. In their study no data on the behaviour of the individual gangliosides under these experimental conditions were given. We have investigated the effect of a drug-induced status epilepticus on the ganglioside concentration and ganglioside pattern in the brain of another species, the rabbit, and would like to present our results briefly.