Upregulation of substance P receptors in angiogenesis associated with chronic airway inflammation in rats
Upregulation of substance P receptors in angiogenesis associated with chronic airway inflammation in rats
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DOI：
10.1152/ajplung.1997.273.3.l565
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发表时间：
1997-09-01
影响因子：
4.9
通讯作者：
McDonald, DM
中科院分区：
文献类型：
--
作者：
Baluk, P;Bowden, JJ;McDonald, DM
In rat airways, substance P released from sensory nerves induces plasma leakage via neurokinin-1 (NK1) receptors on endothelial cells. In pathogen-free rats, both leakage and endothelial NK1 receptors are most abundant in postcapillary venules. In Mycoplasma pulmonis-infected rats, extensive angiogenesis occurs in the tracheal mucosa. The capillary-sized (
