Tetracycline resistance element of pBR322 mediates potassium transport.
Tetracycline resistance element of pBR322 mediates potassium transport.
复制标题
pBR322 的四环素抗性元件介导钾转运。
DOI：
10.1128/jb.160.3.1188-1190.1984
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发表时间：
1984
影响因子：
3.2
通讯作者：
Epstein,W
中科院分区：
文献类型：
--
作者：
Dosch,DC;Salvacion,FF;Epstein,W
质粒pBR 322的四环素抗性元件部分地补充了具有明显受损的K+转运的大肠杆菌K-12突变体的钾转运缺陷。质粒增加K+转运。Tn 10元素不会导致转运增加，表明该效应对于增加四环素抗性的元素并不普遍。
The tetracycline resistance element of plasmid pBR322 partially complements the potassium transport defect of Escherichia coli K-12 mutants having markedly impaired K+ transport. The plasmid increases K+ transport. The Tn10 element does not result in increased transport, demonstrating that the effect is not general for elements that increase resistance to tetracycline.