The relationships between eating disorder pathology and relative reinforcing value of food, delay discounting, and related constructs in adolescents.
The relationships between eating disorder pathology and relative reinforcing value of food, delay discounting, and related constructs in adolescents.
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青少年饮食失调病理学与食物的相对强化价值、延迟折扣和相关结构之间的关系。
DOI：
10.1016/j.appet.2019.104576
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发表时间：
2020
期刊：
影响因子：
5.4
通讯作者：
Temple,JenniferL
中科院分区：
文献类型：
--
作者：
Balantekin,KatherineN;Ziegler,AmandaM;Crandall,AmandaK;Temple,JenniferL
食物是一种主要的食物，在缺乏学习或条件反射的情况下，它会激发行为。食物的相对强化值（RRV）和延迟折扣都与体重状况有关。虽然饮食限制和去抑制已被证明会影响食物的RRV，但有限的工作研究了进食障碍病理学与食物和延迟折扣的RRV之间的关系。因此，本研究的目的是检查进食障碍病理学如何预测食物的RRV、食物消耗的比例、延迟折扣、强化病理学（即，高进食动机和高延迟折扣），以及高能量密集食物（HED;即，每日重复暴露后HED食物的RRV增加）。参与者是167名年龄在12-14岁之间的青少年，他们没有肥胖，参与了一项纵向研究，检查体重变化的预测因素，他们完成了一系列实验室评估，评估了HED食物的RRV，延迟折扣，强化病理学，HED食物的致敏性，以及评估进食障碍病理学的问卷调查。进食障碍病理学与食物的RRV或延迟折扣无关，但确实预测了强化病理学和HED食物的致敏性。当通过体重状况进行探索时，这些关系仅在超重者中观察到。对于体重正常或超重的青少年，没有其他显著的关系。鉴于体重状况似乎缓和了进食障碍病理学和饮食相关结构之间的一些关系，未来的工作应该研究强化病理学和进食障碍病理学如何与体重状况随时间的变化相关。
Food is a primary reinforcer that motivates behavior in the absence of learning or conditioning. Both the relative reinforcing value (RRV) of food and delay discounting are associated with weight status. While dietary restraint and disinhibition have been shown to influence the RRV of food, limited work has examined the relationships between eating disorder pathology and RRV of food and delay discounting. Therefore, the purpose of this study was to examine how eating disorder pathology predicts RRV of food, proportion of food consumed, delay discounting, reinforcement pathology (i.e., high motivation to eat and high delay discounting), and sensitization of high energy dense food (HED; i.e., increases in RRV of HED food after daily repeated exposure). Participants were 167 adolescents ages 12–14 without obesity participating in a longitudinal study examining predictors of weight change who completed a series of laboratory assessments assessing the RRV of food of HED food, delay discounting, reinforcement pathology, sensitization of HED food, and a questionnaire assessing eating disorder pathology. Eating disorder pathology was not related to the RRV of food or delay discounting, but did predict reinforcement pathology and the sensitization of HED food. When explored by weight status, these relationships were only observed for those with overweight. There were no other significant relationships for either adolescents with normal weight or overweight. Given that weight status appeared to moderate some of the relationships between eating disorder pathology and reinforcement-related constructs, future work should examine how reinforcement pathology and eating disorder pathology are related to changes in weight status over time.