Mechanisms linking socioeconomic status to smoking cessation: a structural equation modeling approach.

Mechanisms linking socioeconomic status to smoking cessation: a structural equation modeling approach.
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DOI:
10.1037/a0019285
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发表时间:
2010-05
期刊:
影响因子:
4.2
通讯作者:
Wetter, David W.
Wetter, David W.
中科院分区:
心理学2区
文献类型:
--
作者:
Businelle, Michael S.;Kendzor, Darla E.;Reitzel, Lorraine R.;Costello, Tracy J.;Cofta-Woerpel, Ludmila;Li, Yisheng;Mazas, Carlos A.;Vidrine, Jennifer Irvin;Cinciripini, Paul M.;Greisinger, Anthony J.;Wetter, David W.

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尽管几十年来吸烟率、戒烟率和疾病负担方面一直存在社会经济梯度,但随着时间的推移,这些差异变得更加明显。本研究的目的是开发和测试社会经济地位(SES)与戒烟之间联系机制的概念模型。使用潜变量建模方法对 424 名寻求治疗的吸烟者(34% 非洲裔美国人;33% 拉丁裔;33% 白人)进行了评估。假设的机制包括社会支持、邻里劣势、负面影响/压力、代理和渴望。主要结局是第 4 周的吸烟状况。正如假设的那样,SES 对戒烟有显着的直接和间接影响。具体来说,邻里劣势、社会支持、负面影响/压力和代理介导了社会经济地位与戒烟之间的关系。多组分析表明该模型非常适合跨种族/族裔群体。本研究提出了一种更全面的模型,阐明了 SES 与戒烟之间的具体机制。针对低 SES 吸烟者并解决当前模型中确定的具体途径的政策、社区和个人干预措施可能会减弱 SES 对戒烟的影响。
Although there has been a socioeconomic gradient in smoking prevalence, cessation, and disease burden for decades, these disparities have become even more pronounced over time. The aim of the current study was to develop and test a conceptual model of the mechanisms linking socioeconomic status (SES) to smoking cessation. The conceptual model was evaluated using a latent variable modeling approach in a sample of 424 smokers seeking treatment (34% African American; 33% Latino; 33% White). Hypothesized mechanisms included social support, neighborhood disadvantage, negative affect/stress, agency, and craving. The primary outcome was week 4 smoking status. As hypothesized, SES had significant direct and indirect effects on cessation. Specifically, neighborhood disadvantage, social support, negative affect/stress, and agency mediated the relation between SES and smoking cessation. A multiple group analysis indicated that the model was a good fit across racial/ethnic groups. The present study yielded one of the more comprehensive models illuminating the specific mechanisms that link SES and smoking cessation. Policy, community, and individual-level interventions that target low SES smokers and address the specific pathways identified in the current model could potentially attenuate the impact of SES on cessation.
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