12‐HETE promotes late‐phase responses in a murine model of allergic rhinitis
12‐HETE promotes late‐phase responses in a murine model of allergic rhinitis
复制标题
12-HETE 促进过敏性鼻炎小鼠模型的晚期反应
DOI：
10.1111/all.15547
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发表时间：
2022
期刊：
影响因子：
12.4
通讯作者：
Murata Takahisa
中科院分区：
文献类型：
--
作者：
Nakamura Tatsuro;Tachibana Yuri;Murata Takahisa
虽然这些免疫细胞产生几种类型的脂质介质，[2]脂质介质的类型和作用在晚期仍然未知。变应性鼻炎（AR）是最常见的慢性上呼吸道疾病之一。12-HETE促进变应性鼻炎小鼠模型的晚期反应。[摘自文章]过敏的版权是威利-布莱克威尔的财产，其内容不得复制或通过电子邮件发送到多个网站或张贴到一个列表服务器未经版权保持器的明确书面许可。但是，用户可以打印、下载或通过电子邮件发送文章供个人使用。这篇摘要可以删节。对副本的准确性不作任何保证。用户应参考材料的原始出版版本以获取完整摘要。版权适用于所有摘要。
Although these immune cells produce several types of lipid mediators,[2] the types and roles of lipid mediators act on late-phase remain unknown. Allergic rhinitis (AR) is amongst the most common chronic upper respiratory diseases. 12-HETE promotes late-phase responses in a murine model of allergic rhinitis.[Extracted from the article]Copyright of Allergy is the property of Wiley-Blackwell and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. Copyright applies to all Abstracts.