Persistent Stapedial Artery and Chronic Otitis Media
Persistent Stapedial Artery and Chronic Otitis Media
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持续性镫骨动脉和慢性中耳炎
DOI:
10.1067/mhn.2000.109931
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发表时间:
2000
期刊:
影响因子:
--
通讯作者:
A. Antonelli
中科院分区:
文献类型:
--
作者:
D. Zanetti;C. Piazza;A. Antonelli
tis media with hearing loss and purulent otorrhea, associated with a persistent pulsatile tinnitus in the right ear that had been present from birth. The last symptom had been previously attributed to the middle ear disease. Her medical and family history were otherwise unremarkable. A pure-tone audiogram showed moderate bilateral conductive hearing loss in the lowfrequency range. Otoscopic examination showed a large pulsatile vessel through a right subtotal tympanic membrane perforation. Its course and relationships with adjacent anatomic structures were suggestive of a persistent stapedial artery (PSA). A high-resolution CT scan of the temporal bones was therefore performed to rule out middle or inner ear malformations, often associated with this finding.1 Radiologic examination was negative in this respect and indirectly confirmed the presence of a PSA by enlargement of the tympanic portion of the right facial canal close to the geniculate ganglion. Absence of the foramen spinosum on the right indicated a lack of normal middle meningeal artery branching from the internal maxillary, consistent with a diagnosis of PSA.2 No cholesteatoma was detected, and there was no clinical indication for an angiography. Micro-otoscopy allowed visualization of the complete course of the vessel toward the intracranial compartment. An underlay myringoplasty with autologous tragal perichondrium was performed with the patient under general anesthesia. During surgery the vessel was found to be 2 mm in diameter, coursing from the floor of the hypotympanum across the promontory and between the stapedial crura (Fig 1). The artery entered the facial canal just behind the cochleariform process. No stapedial ankylosis or ossicular malformations were seen. Any surgical maneuver on the aberrant artery was avoided to minimize the risks of profuse bleeding and neurologic sequelae.1 The postoperative course was uneventful, and at 1-year follow-up the right neotympanum was intact and no recurrence of the inflammatory process was detected. The patient reported no subjective change of the tinnitus. A myringoplasty was then performed in the opposite ear, where no anatomic abnormalities were observed.