The type III secretion effector xopXccN of Xanthomonas campestris pv. campestris is required for full virulence
The type III secretion effector xopXccN of Xanthomonas campestris pv. campestris is required for full virulence
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DOI：
10.1016/j.resmic.2007.12.004
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发表时间：
2008-04-01
影响因子：
2.6
通讯作者：
Tang, Ji-Liang
中科院分区：
文献类型：
--
作者：
Jiang, Bo-Le;He, Yong-Qiang;Tang, Ji-Liang
XopN最初从野油菜黄单胞菌致病变种（Xanthomonascampestris pv.）vesicatoria作为效应子通过III型分泌系统（T3SS）易位到植物细胞中，并且是致病性所需的。我们在这里报告，xopN同源物在X。野油菜致病变种campestris基因组，命名为xopXccN，也编码T3SS效应子，并且是完全毒力所必需的。我们进一步证明xopXccN的表达受到关键hrp（过敏反应和致病性）调节因子HrpG和HrpX的正调控。(C)2008年由Elsevier Masson SAS出版。
XopN was originally identified from Xanthomonas campestris pv. vesicatoria as an effector translocated into plant cells via the type III secretion system (T3SS), and is required for pathogenicity. We report here that the xopN homologue in the X. campestris pv. campestris genome, named xopXccN, also encodes a T3SS effector and is required for full virulence. We further demonstrate that expression of xopXccN is positively regulated by the key hrp (hypersensitive response and pathogenicity) regulators HrpG and HrpX. (C) 2008 Published by Elsevier Masson SAS.