FoxP2 and Vocalization

FoxP2 and Vocalization
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FoxP2 和发声

DOI:
10.1075/slcs.144.09whi
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发表时间:
2013
影响因子:
5.8
通讯作者:
S. White
S. White
中科院分区:
医学2区
文献类型:
--
作者:
S. White

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从语言学家的角度来看,发出新声音的能力似乎不是语言的关键组成部分。然而,当这种能力受损时,受影响的个人的社会和情感后果可能是严重的,正如那些患有发育或损伤相关的言语障碍的人所证明的那样。我们如何理解这种声音学习特征,以及在语言进化的框架中应该把它放在哪里?在这里,我认为,研究在声乐学习障碍的影响支持大脑通路是一个很好的起点。由于这种研究在很大程度上仅限于人类的非侵入性方法,因此调查其他具有这种罕见特征的动物为比较分析发声学习(包括人类语音)的分子,细胞和突触基础铺平了道路。这种调查可以突出共同的进化途径以及关键的弯路。
From a linguist’s point of view, the ability to vocalize new sounds may not seem to be a critical component of language. Yet when this ability is impaired, the social and emotional consequences for the affected individual can be severe, as evidenced by those suffering from developmental or injury-related speech disorders. How are we to understand this vocal learning trait, and where should it be placed within a framework for language evolution? Here, I argue that studying the supporting brain pathways that are affected in vocal learning disorders is a good place to start. Since such study is largely limited to noninvasive methods in humans, investigating other animals that possess this rare trait paves the way for a comparative analysis of the molecular, cellular, and synaptic bases of vocal production learning, including human speech. This kind of inquiry can highlight shared evolutionary pathways as well as key detours.
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发表时间: 2008-03-11
期刊: CURRENT BIOLOGY
影响因子: 9.2
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