Autonomic arousals related to traffic noise during sleep
Autonomic arousals related to traffic noise during sleep
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DOI：
10.1093/sleep/31.4.569
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发表时间：
2008-04-01
期刊：
影响因子：
5.6
通讯作者：
Basner, Mathias
中科院分区：
文献类型：
--
作者：
Griefahn, Barbara;Broede, Peter;Basner, Mathias
目的：目的：分析交通噪声对睡眠中心率（HR）的影响，以及声学参数、夜间时间和瞬时睡眠阶段对这些反应的影响（19 - 28岁）。测量和结果：参与者在连续3周的每一周中连续4个晚上睡在实验室里，并暴露在飞机、道路、或轨道交通噪音，每周排列。每周的4个夜晚包括一个安静的夜晚（32 dBA）和3个夜晚的随机序列，在此期间，飞机，铁路交通或道路交通噪音发生的最大水平为45 - 77 dBA。记录所有夜间的多导睡眠图和心电图。在觉醒的情况下，HR改变包括持续> 1分钟的单相升高，平均最大HR升高为30 bpm。虽然很明显是由噪音事件触发的，但唤醒本身而不是声学参数决定了反应的程度和模式。在没有觉醒的情况下，HR反应是双相的，包括初始加速，最大HR升高约9 bpm，随后减速低于基线。这些变化明显的声学参数（交通模式，最大水平，上升率）的影响，以及由瞬时睡眠stage.Conclusions：心脏反应没有习惯于交通噪声在夜间，因此可能发挥关键作用，促进交通噪声引起的心血管疾病。如果是这样的话，这些后果更有可能伴随着觉醒的反应比没有觉醒的情况。
Aim: To analyze the heart rate (HR) response to traffic noise during sleep and the influence of acoustic parameters, time of night, and momentary sleep stage on these responses.Participants: Twelve women and 12 men (19-28 years).Measurements and Results: The participants slept in the laboratory for 4 consecutive nights in each of 3 consecutive weeks and were exposed to aircraft, road, or rail traffic noise with weekly permutations. The 4 nights of each week consisted of a random sequence of a quiet night (32 dBA) and 3 nights during which aircraft, rail traffic, or road traffic noises occurred with maximum levels of 45-77 dBA. The polysomnogram and the electrocardiogram were recorded during all nights. In case of awakenings, the HR alterations consisted of monophasic elevations for >1 min, with mean maximum HR elevations of 30 bpm. Though obviously triggered by the noise events, the awakenings per se rather than the acoustical parameters determined the extent and pattern of the response. Without awakenings, HR responses were biphasic and consisted of initial accelerations with maximum HR elevations of about 9 bpm followed by decelerations below the baseline. These alterations were clearly influenced by the acoustic parameters (traffic mode, maximum level, rate of rise) as well as by the momentary sleep stage.Conclusions: Cardiac responses did not habituate to traffic noise within the night and may therefore play a key role in promoting traffic noise induced cardiovascular disease. If so, these consequences are more likely for responses accompanied by awakenings than for situations without awakenings.