Protection of CNS neurons by Heat Shock Proteins

热休克蛋白对中枢神经系统神经元的保护

• 批准号: 196407
• 负责人: Mearow Karen M
• 金额: $ 11.92万
• 依托单位: Memorial University of Newfoundland
• 依托单位国家: 加拿大
• 项目类别: Operating Grants
• 财政年份: 2009
• 资助国家: 加拿大
• 起止时间: 2009-09-01 至 2012-09-01
• 项目状态: 已结题
• 来源: https://webapps.cihr-irsc.gc.ca/decisions/p/project_details.html?applId=196407&lang=en
• 关键词: Biochemical And Cellular Analyses; Cell Culture Of Cns Neurons; Cytoskeleton And Axonal Transport; Imaging And Confocal Microscopy; Neuronal Toxicity; Protein-Protein Interactions

Alzheimer's disease is thought to be caused by cumulative effects of beta-amyloid that can result in structures in the brain known as plaques and tangles. The plaques often consist of beta amyloid, as well as other proteins that can include those that ar

阿尔茨海默病被认为是由β -淀粉样蛋白的累积效应引起的，β -淀粉样蛋白会导致大脑中的斑块和缠结结构。斑块通常由-淀粉样蛋白以及其他蛋白质组成，其中包括淀粉样蛋白