Modeling the mechanism of translation control of ATF4 by upstream open reading frames under normal and stressful physiological conditions.
Modeling the mechanism of translation control of ATF4 by upstream open reading frames under normal and stressful physiological conditions.
批准号:
364317
负责人:
Marasco Olivia N
金额:
$0.36万
依托单位国家:
加拿大
项目类别:
Studentship Programs
财政年份:
2017
资助国家:
加拿大
项目状态:
已结题
起止时间:
2017-01-01 至 2017-05-01
中文摘要
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英文摘要
ATF4 is a regulatory protein involved in the cellular stress response in mammals. Expression of ATF4 is normally repressed, but under stress conditions, ATF4 is expressed, and turns on the transcription of several other genes required to handle the stress
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