Plant priming in biotic stress responses mediated by CDPK signalling (C02)

CDPK 信号介导的植物启动生物胁迫反应 (C02)

• 批准号: 223429309
• 金额: --
• 依托单位: Abteilung Biochemie pflanzlicher Interaktionen
• 依托单位国家: 德国
• 项目类别: Collaborative Research Centres
• 财政年份: 2012
• 资助国家: 德国
• 起止时间: 2011-12-31 至 2020-12-31
• 项目状态: 已结题
• 来源: https://gepris.dfg.de/gepris/projekt/223429309?language=en
• 关键词: Plant; priming; biotic; stress; responses

Complementary to a role of calcium-dependent protein kinase-(CDPK)-mediated signalling in early local immune responses of Arabidopsis thaliana to phytopathogens, project C2 studies the function of CDPKs in priming of long-term, systemic immune responses (systemic acquired resistance; SAR). We characterised CPK5 as major positive regulator of local and systemic immune responses, and thus, as a key regulator in immune priming. Future research will address biochemical, post-translational enzyme modifications, providing a mechanistic understanding of priming (and forgetting), and CPK5-mediated control of transcription factor-dependent reprogramming in gene expression during SAR.

除了钙依赖蛋白激酶(CDPK)介导的信号转导在拟南芥对植物病原体的早期局部免疫反应中的作用之外，C2项目研究了CDPKs在启动长期的系统性免疫反应(系统获得性抗性；SAR)中的功能。我们认为CPK5是局部和全身免疫反应的主要正向调节因子，因此是免疫启动的关键调节因子。未来的研究将涉及生化、翻译后酶修饰，提供对启动(和遗忘)的机制理解，以及CPK5介导的对SAR期间转录因子依赖的基因表达重新编程的控制。