Genetical, physiological and ecological studies on the mechanisms of insecticide resistance.

在褐飞虱体内，马拉硫磷与氰戊菊酯之间存在负相关的交叉抗性现象。在马拉硫磷和乙基苯丙胺中也发现了类似的现象。在马拉硫磷抗性品系中，β&lt；14&gt；C-马拉硫磷的角质层渗透被抑制，而其代谢被增强。在小菜蛾中，苯乙醇酯抗性是由于其表皮渗透率低和乙酰胆碱酯酶对苯乙醇胺的敏感性降低所致。苯硫磷的遗传受不完全显性因素控制。小菜蛾对氰戊菊酯的抗性机制是由于其表皮穿透性降低和神经系统对氰戊菊酯的敏感性降低所致。氰戊菊酯抗性的遗传受不完全隐性因素控制。

In the brown planthopper, Nilaparvata leugens, the phenomenon of negatively corelated cross resistance was found between malathion and fenvalerate. The similar phenomenon was also found between malathion and ethophenprox. In malathion resistant strain, the cuticular penetration of ^<14>C-malathion was found to be redyced and the metabolism of ^<14>C-malathion was found to be enhanced. However, there were no differences in cuticular penetration, metabolism and binding of ^<14>C-fenvalerate to nerve preparation.In the diamondback moth, Plutella xylostella, phenthoate resistance was found to be caused by low cuticular penetration of ^<14>C-phenthoate and reduced sensitivity of acetylcholinesterase to phenthoate-oxon. Inheritance of phenthoate was controled by an incomplete dominant factor. The resistance mechanism of fenvalerate in the diamondback moth was caused by reduced cuticular penetration of ^<14>C-fenvalerate and reduced sensitivity of nervous system to fenvalerate. The inheritance of fenvalerate resistance was considered to be controled by and imcomplete recessive factor.