Calcium metabolism in normal pregnancy and pregnancy induced hypertension

因为怀孕在交货后迅速地提高了高血压，所以我想这是由于一种分离在体外循环中造成的。对内皮细胞的伤害是由血清引起的，它可能在怀孕诱导的高血压中发挥作用。We investigated the relation between serum samples from women with pregnancy induced hypertension and endothelial cell Cas^\+\。Ca^<++>是由血清样本从女性怀孕诱导的高血压引起的。我们相信血清样本中的一些因素与女性怀孕诱导的高血压可以调节由内皮细胞引起的硝酸氧化物的周期性保密。然后，我们研究了一种天然气晶体在泌尿系统流动速度波形式中的基本生物学特征。我们五个怀孕的人都在16-17周的时间里使用了这项研究。微珠子（Gelfoam） were stepwisely administered into the uterine artery and changes in uterine circulation were assessed by Doppler velocimetry。脉冲性指数线性 ... More 与紫外线抗压力相关，并提供了一个高度的相关性。当肠道抗压力和PI出现在2.13±和450±92 mmHg·min·L^<-1>时，对此。它可以包括PI的提升和泌尿系统中的diastolic notes的存在，是高泌尿系统血管阻力和有缺陷的泌尿系统循环的指标。Furthermore，我们调查了正常怀孕和怀孕诱导性高血压中的初级酸水平。这些水平是用高性能液体色谱法测量的，带有柱切换。正常怀孕者个人在20和30周内展示了一个有意义的怀孕高峰，与第一次怀孕的那些相比。在硝酸氧化物生产期间，正常怀孕可能导致一种生理性升高的肾上腺素在硝酸氧化物生产中的高消耗。怀孕诱导的高血压中的缺失可能与硝酸氧化物生产中的降解有关。Less（低）

Because pregnancy induced hypertension improves rapidly after delivery, it is thought to be caused by a disturbance in uteroplacental circulation. Injury to endothelial cells caused by serum may have a role in the onset of pregnancy induced hypertension. We investigated the relation between serum samples from women with pregnancy induced hypertension and endothelial cell Cas^<++>. The Ca^<++> was induced by serum samples from women with pregnancy induced hypertension. We believe that some factors in serum samples from women with pregnancy induced hypertension could modulate periodic secretion of nitric oxide by endothelial cells. Then, we investigated the basic biological significance of diastolic notches in uterine artery flow velocity waveforms. Five pregnant ewe at 16-17 weeks of pregnancy were used in the study. Micro-beads (Gelfoam) were stepwisely administered into the uterine artery and changes in uterine circulation were assessed by Doppler velocimetry. Pulsatility index linear … More ly correlated with uterine vascular resistance, giving a high correlation coefficient. The mean values of uterine vascular resistance and PI when diastolic notches appeared were 2.13±and 450±92 mmHg・min・L^<-1>, respectively. It could be concluded that the elevation of PI and presence of diastolic notches in the uterine arteries are indicators of high uterine vascular resistance and impaired uterine circulation. Furthermore, we investigated the urinary levels of orotic acid in normal pregnancy and in pregnancy-induced hypertension. Such levels were measured using high-performance liquid chromatography with column-switching. Normal pregnant individually showed a significant elevation at 20 and 30 weeks of gestation as compared with those in the first trimester. A high consumption of arginine in nitric oxide production during normal pregnancy may cause a physiological elevation of urinary orotic acid. The absence of an elevation in pregnancy-induced hypertension may be associated with a decrease in nitric oxide production. Less