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HHV-6 IN AIDS--MECHANISMS OF CD4 GENE ACTIVATION

HHV-6 IN AIDS--MECHANISMS OF CD4 GENE ACTIVATION
艾滋病中的HHV-6--CD4基因激活机制
批准号：
2423531
负责人：
MARVIN S REITZ
金额：
$7.25万
依托单位：
UNIV OF MARYLAND, COLLEGE PARK
依托单位国家：
美国
项目类别：
财政年份：
1997
资助国家：
美国
项目状态：
已结题
起止时间：
1997-08-01 至 1999-07-31
来源：
https://reporter.nih.gov/project-details/2423531
关键词：
AIDS CD4 molecule Herpesviridae disease complementary DNA gene induction /repression genetic enhancer element genetic promoter element host organism interaction human herpesvirus 6 immunogenetics microorganism culture microorganism immunology molecular cloning nucleic acid sequence transcription factor virus genetics virus protein

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DESCRIPTION (adapted from applicant's abstract): The investigators propose to study the interactions between HHV-6 and genetic elements controlling CD4 gene expression by examining: 1) which genetic elements (promoter, enhancer, silencer) are activated/derepressed by HHV-6, and 2) which HHV-6 gene(s) is(are) responsible for turning on the CD4 gene. This knowledge may shed light on the immunobiological properties of HHV-6 and how its activity may contribute to HIV disease, and it may also contribute to a more complete understanding of fundamental immunological questions such as the control of gene expression of immunocompetent cells.
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Effects of Ritonavir on HHV-8 vGPCR signaling and tumorigenesis
  • 批准号：
    7491371
  • 项目类别：
  • 资助金额：
    $13.84万
  • 财政年份：
    2006
  • 负责人：
    MARVIN S REITZ
  • 依托单位：
    UNIVERSITY OF MARYLAND BALTIMORE
Effects of Ritonavir on HHV-8 vGPCR signaling and tumorigenesis
Pathogenic Mechanisms of HHV-8 ORF74
Pathogenic Mechanisms of HHV-8 ORF74
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