CELLULAR IMMUNE MECHANISMS OF HOST RESPONSE TO HIV

宿主对 HIV 反应的细胞免疫机制

基本信息

项目摘要

Dr. Herve Sroussi, who is integrating his PhD studies in oral biology with specialty training in oral medicine, was appointed to this institutional DSA in July of 1998. Dr. Sroussi, who received his DMD in 1994 from Hebrew University in Israel, came to California as a permanent resident in June of 1996, and engaged in research and clinical activities under the supervision of oral medicine and oral biology faculty during 1997, leading to his entrance into our DSA program with the oral biology-oral medicine combination. This particular track permits full integration of both components from the start, due to the flexibility inherent in our oral medicine program, which we anticipate will result in a shorter time to completion. Dr. Sroussi's research interests are currently focused on cellular immune mechanisms of the host response to HIV infection, and co-infections with other viruses, particularly the human papillomavirus (HPV). Kl6DE003860005 Dr. Susan Hyde, who is pursuing her PhD in epidemiology in combination with specialty training in dental public health, was appointed to this institutional DSA in July of 1998. Dr. Hyde received her DDS from UCSF in 1992 and her MPH from UC Berkeley in 1998, and began her DSA program last July. Her focus this year has been mainly on her dental public health residency and developing her research skills in epidemiology under the auspices of Dr. Jane Weintraub. She is making very good progress in both areas and will begin her intensive PhD work in epidemiology at Berkeley starting this summer, while continuing her public health residency experiences at a reduced level of commitment over the next several years.
埃尔韦斯鲁西博士,谁是整合他的博士研究在口腔生物学 在口腔医学专业培训,被任命为这个 1998年7月机构每日生活津贴。 Sroussi博士在1999年接受了DMD, 1994年从以色列希伯来大学毕业,来到加州, 1996年6月住院,从事科研和临床工作, 在口腔医学和口腔生物学的监督下开展活动 1997年,他进入了我们的DSA课程, 口服生物制剂-口服药物组合物。 这条特别的赛道 允许从一开始就完全集成两个组件, 我们的口腔医学计划固有的灵活性,我们预计 将导致更短的完成时间。 斯鲁西博士的研究 目前的兴趣集中在细胞免疫机制, 宿主对艾滋病毒感染的反应,以及与其他病毒的合并感染, 特别是人乳头瘤病毒(HPV)。 电话:+86-0516 - 8888888 博士苏珊·海德正在攻读流行病学博士学位, 在牙科公共卫生专业培训,被任命为这 1998年7月机构每日生活津贴。 海德博士从加州大学旧金山分校获得了她的DDS 1992年毕业,1998年获得加州大学伯克利分校公共卫生硕士学位,并开始了她的DSA课程 去年七月。 她今年的重点主要是她的牙科公众 卫生住院医师和发展她的流行病学研究技能 由简·温特劳布博士主持 她做得很好 在这两个领域的进展,并将开始她密集的博士工作, 流行病学在伯克利开始今年夏天,同时继续她的 降低承诺水平的公共卫生住院经验 在接下来的几年里。

项目成果

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Herve Y Sroussi其他文献

Herve Y Sroussi的其他文献

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{{ truncateString('Herve Y Sroussi', 18)}}的其他基金

Long Term Oral Health Outcomes in the Chronic GVHD Consortium
慢性 GVHD 联盟的长期口腔健康结果
  • 批准号:
    10221670
  • 财政年份:
    2019
  • 资助金额:
    $ 6.02万
  • 项目类别:
Long Term Oral Health Outcomes in the Chronic GVHD Consortium
慢性 GVHD 联盟的长期口腔健康结果
  • 批准号:
    10664053
  • 财政年份:
    2019
  • 资助金额:
    $ 6.02万
  • 项目类别:
Long Term Oral Health Outcomes in the Chronic GVHD Consortium
慢性 GVHD 联盟的长期口腔健康结果
  • 批准号:
    10455702
  • 财政年份:
    2019
  • 资助金额:
    $ 6.02万
  • 项目类别:
CALPROTECTIN AS A MOLECULAR SENSOR AND REGULATOR OF OXIDATION IN WOUND HEALING
钙卫蛋白作为伤口愈合中的分子传感器和氧化调节剂
  • 批准号:
    7840990
  • 财政年份:
    2009
  • 资助金额:
    $ 6.02万
  • 项目类别:
CALPROTECTIN AS A MOLECULAR SENSOR AND REGULATOR OF OXIDATION IN WOUND HEALING
钙卫蛋白作为伤口愈合中的分子传感器和氧化调节剂
  • 批准号:
    7933267
  • 财政年份:
    2009
  • 资助金额:
    $ 6.02万
  • 项目类别:
CALPROTECTIN AS A MOLECULAR SENSOR AND REGULATOR OF OXIDATION IN WOUND HEALING
钙卫蛋白作为伤口愈合中的分子传感器和氧化调节剂
  • 批准号:
    7442159
  • 财政年份:
    2006
  • 资助金额:
    $ 6.02万
  • 项目类别:
CALPROTECTIN AS A MOLECULAR SENSOR AND REGULATOR OF OXIDATION IN WOUND HEALING
钙卫蛋白作为伤口愈合中的分子传感器和氧化调节剂
  • 批准号:
    7258384
  • 财政年份:
    2006
  • 资助金额:
    $ 6.02万
  • 项目类别:
CALPROTECTIN AS A MOLECULAR SENSOR AND REGULATOR OF OXIDATION IN WOUND HEALING
钙卫蛋白作为伤口愈合中的分子传感器和氧化调节剂
  • 批准号:
    7146264
  • 财政年份:
    2006
  • 资助金额:
    $ 6.02万
  • 项目类别:
CALPROTECTIN AS A MOLECULAR SENSOR AND REGULATOR OF OXIDATION IN WOUND HEALING
钙卫蛋白作为伤口愈合中的分子传感器和氧化调节剂
  • 批准号:
    7637757
  • 财政年份:
    2006
  • 资助金额:
    $ 6.02万
  • 项目类别:
CELLULAR IMMUNE MECHANISMS OF HOST RESPONSE TO HIV
宿主对 HIV 反应的细胞免疫机制
  • 批准号:
    6338738
  • 财政年份:
    2000
  • 资助金额:
    $ 6.02万
  • 项目类别:

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阐明胰腺癌免疫逃避的分子和细胞机制
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