Increasing fitness potential of plants through chromatin landscape analysis during stress responses

缺水条件下的胁迫反应被诱导，使生存和同时芽生长减少，以限制水分流失。因此，研究高等植物逆境响应和逆境耐受的分子机制，对提高植物的逆境生存能力具有重要意义。脱落酸（阿坝）和茉莉酸（JAs）胁迫激素信号通路之间的相互作用是植物生长与环境相适应的核心。JAs引起了一个短暂的增长停滞在一个矛盾的，所谓的，准备去的状态，其次是重置的增长率。茉莉酸甲酯（MeJA）和阿坝水平在干旱胁迫期间增加，MeJA在干旱诱导的产量损失中起重要作用。引发或硬化的过程涉及生物或非生物胁迫的先前经历，使植物对未来的暴露更具抗性。染色质重塑是调节环境反应基因的重要因素。本项目将研究植物记忆胁迫信息的分子机制，以克服生物胁迫和非生物胁迫对农业生产的影响。

Stress responses under water scarcity are induced to enable survival and simultaneously shoot growth is reduced to limit water loss. It is therefore of paramount importance to analyze the molecular mechanisms regulating stress response and tolerance in higher plants to enhance stress-survival. The interplay between abscisic acid (ABA) and jasmonates (JAs) stress hormone signalling pathways is central to align plant growth with the environment. JAs evokes a transient growth arrest in an ambivalent, so called, READY-TO-GO state, followed by the reset of growth rate. Methyl jasmonate (MeJA) and ABA levels increase during drought stress and MeJA plays an important role in drought-induced loss of grain yield. The process of priming or hardening involves previous experience of a biotic or an abiotic stress making a plant more resistant to future exposure. Chromatin remodelling is emerging as an important factor to regulate environmental response genes. Exposure to a priming agent could modify an epigenetic mark such histone modification, enhancing responses to subsequent stress.This project will establish the molecular mechanism by which plants memorize information on stress exposure to overcome the limits that biotic and abiotic stresses impose on agricultural production due to down-regulation of yield.