Bi-Annual Meeting of the IUIS Expert Committee on PID
Bi-Annual Meeting of the IUIS Expert Committee on PID
批准号:
7280473
负责人:
RAIF SALIM GEHA
金额:
$0.0万
依托单位国家:
美国
项目类别:
财政年份:
2005
资助国家:
美国
项目状态:
已结题
起止时间:
2005-05-15 至 2010-04-30
中文摘要
原发性免疫缺陷疾病(PID)提供了一个很好的窗口进入免疫系统的功能。在过去的15年中,已经阐明了100多个PID的分子基础。国际免疫学会联合会(IUIS) PID专家委员会每两年召开一次会议,目的是向从事PID工作的科学家和医生通报基础和临床科学领域的最新进展,这些进展对PID患者的调查、诊断和护理有影响。会议还有助于刺激pid领域的临床试验,包括开发新的治疗策略。
英文摘要
Primary immune deficiency diseases (PID) provide an excellent window into the functioning of the immune system. During the past 15 years the molecular basis of more than 100 PID has been elucidated. The purpose of the bi-annual meeting of the International Union of Immunological Societies (IUIS) Expert Committee on PID is to update the community of scientists and physicians who work in PID to advances in the field of basic and clinical science that have impact on the investigation, diagnosis and care of patients with PID. The meeting also serves to stimulate clinical trials in the field of PIDs, including the development of novel therapeutic strategies.
The meeting is structured over 3 days. The first 2.5 days consist of approximately 5 scientific sessions, in which basic science presentations of cutting edge research in immunology are followed by presentations describing novel gene defects or new understandings of the structure and function of previously described genes deficient in patients with PID or new therapies for PID. There are normally a total of about 30 speakers. The Faculty includes distinguished scientists who have significantly contributed to advances in molecular
and cellular immunology, biochemistry, and cell signaling and of translational researchers who have made advances in the pathophysiology diagnosis and treatment of PID during the past two years. The meeting is attended by approximately 100-120 invited physicians and scientists who are actively engaged in PID research and patient care all over the world.The scientific sessions are followed by a half day meeting of the Committee of Experts on PID to update the classification of PID. During this meeting the classification tables of PID from the previous meeting are updated the published in ahigh impact immunology journal. We are seeking support for three meetings to be held in 2005, 2007 and 2009. The 2005 meeting will be held June 17-19 in Budapest, Hungary.
期刊论文(0)
专著(0)
科研奖励(0)
会议论文
Mechanisms of enhanced food allergy by S. aureus skin colonization in Atopic Dermatitis
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批准号:10638821
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项目类别:
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资助金额:$80.01万
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财政年份:2023
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负责人:RAIF SALIM GEHA
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依托单位:
Molecular and cellular mechanisms in food anaphylaxis
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批准号:10408011
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项目类别:
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资助金额:$54.8万
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财政年份:2020
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负责人:RAIF SALIM GEHA
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依托单位:
Mechanisms of a Novel Combined Immunodeficiency Caused by a Homozygous Mutation in COPG1
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批准号:10265627
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项目类别:
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资助金额:$44.25万
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财政年份:2020
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负责人:RAIF SALIM GEHA
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依托单位:
Molecular and cellular mechanisms in food anaphylaxis
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批准号:10030396
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项目类别:
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资助金额:$54.8万
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财政年份:2020
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负责人:RAIF SALIM GEHA
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依托单位:
Genetic and microbial modifiers of Atopic Dermatitis (AD): Mechanisms of increased AD severity in patients with the R576 polymorphism in IL-4Ra and impact of S aureus skin decolonization on AD
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批准号:10589788
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项目类别:
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资助金额:$52.65万
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财政年份:2020
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负责人:RAIF SALIM GEHA
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依托单位:
Mechanisms of a Novel Combined Immunodeficiency Caused by a Homozygous Mutation in COPG1
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批准号:10159668
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项目类别:
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资助金额:$38.98万
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财政年份:2020
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负责人:RAIF SALIM GEHA
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依托单位:
Genetic and microbial modifiers of Atopic Dermatitis (AD): Mechanisms of increased AD severity in patients with the R576 polymorphism in IL-4Ra and impact of S aureus skin decolonization on AD
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批准号:10381494
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项目类别:
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资助金额:$52.65万
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财政年份:2020
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负责人:RAIF SALIM GEHA
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依托单位:
Genetic and microbial modifiers of Atopic Dermatitis (AD): Mechanisms of increased AD severity in patients with the R576 polymorphism in IL-4Ra and impact of S aureus skin decolonization on AD
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批准号:9974923
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项目类别:
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资助金额:$53.07万
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财政年份:2020
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负责人:RAIF SALIM GEHA
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依托单位:
Mechanisms of a Novel Combined Immunodeficiency Caused by a Homozygous Mutation in COPG1
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批准号:10493663
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项目类别:
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资助金额:$5.27万
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财政年份:2018
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负责人:RAIF SALIM GEHA
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依托单位:
Mechanisms of a Novel Combined Immunodeficiency Caused by a Homozygous Mutation in COPG1
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批准号:10394995
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项目类别:
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资助金额:$44.25万
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财政年份:2018
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负责人:RAIF SALIM GEHA
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依托单位:
Mechanisms of a Novel Combined Immunodeficiency Caused by a Homozygous Mutation in COPG1
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批准号:9912718
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项目类别:
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资助金额:$44.25万
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财政年份:2018
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负责人:RAIF SALIM GEHA
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依托单位:
Mechanisms of disease in patients with I?B? mutations
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批准号:9335262
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项目类别:
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资助金额:$26.55万
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财政年份:2016
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负责人:RAIF SALIM GEHA
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依托单位:
Mechanisms of food allergy elicited by cutaneous sensitization
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批准号:9755337
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项目类别:
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资助金额:$44.25万
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财政年份:2016
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负责人:RAIF SALIM GEHA
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依托单位:
Mechanisms of disease in patients with I?B? mutations
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批准号:9090492
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项目类别:
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资助金额:$22.13万
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财政年份:2016
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负责人:RAIF SALIM GEHA
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依托单位:
Role of Myeloid Derived Suppressor Cells in intestinal inflammation
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批准号:8772887
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项目类别:
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资助金额:$26.38万
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财政年份:2014
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负责人:RAIF SALIM GEHA
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依托单位:
Role of Myeloid Derived Suppressor Cells in intestinal inflammation
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批准号:8898001
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项目类别:
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资助金额:$22.1万
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财政年份:2014
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负责人:RAIF SALIM GEHA
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依托单位:
Novel Immunodeficiency caused by TFRC Mutation
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批准号:8726281
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项目类别:
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资助金额:$21.75万
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财政年份:2013
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负责人:RAIF SALIM GEHA
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依托单位:
Novel Immunodeficiency caused by TFRC Mutation
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批准号:8564631
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项目类别:
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资助金额:$24.53万
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财政年份:2013
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负责人:RAIF SALIM GEHA
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依托单位:
Role of DOCK8 in B Cell Function
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批准号:8821572
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项目类别:
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资助金额:$44.13万
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财政年份:2013
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负责人:RAIF SALIM GEHA
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依托单位:
Role of DOCK8 in B Cell Function
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批准号:8504207
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项目类别:
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资助金额:$41.13万
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财政年份:2013
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负责人:RAIF SALIM GEHA
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依托单位: