Role of SAA in Inflammation and Immunity

SAA 在炎症和免疫中的作用

• 批准号: 8089973
• 负责人: RICHARD D YE
• 金额: $ 40.63万
• 依托单位: UNIVERSITY OF ILLINOIS AT CHICAGO
• 依托单位国家: 美国
• 财政年份: 1997
• 资助国家: 美国
• 起止时间: 1997-07-01 至 2011-07-31
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/8089973
• 关键词: A Mouse; Abbreviations; Acute; Acute-Phase Proteins; Acute-Phase Reaction; Advanced Glycosylation End Products; Amyloid; Anti-Inflammatory Agents; Anti-inflammatory; Apolipoprotein A-I; Apolipoprotein E; Apolipoproteins; Arthritis; Atherosclerosis; Autoimmune Diseases; Biological Markers; Biological Process; Blood Circulation; Bone Marrow; Burn Trauma; C-reactive protein; CD36 gene; CSF3 gene; Cell Line; Cells; Clinical; Collagen Arthritis; DNA; Development; Disease; Disease Progression; Disease model; Fibroblasts; Funding; G Protein-Coupled Receptor Genes; G-Protein-Coupled Receptors; GTP-Binding Proteins; Genetic; Glutathione S-Transferase; Goals; Hepatic; Hepatocyte; High Density Lipoproteins; Human; Immune; Immunity; Individual; Infection; Infectious Agent; Inflammation; Inflammation Mediators; Inflammatory; Intra-Articular Injections; Investigation; Knockout Mice; Knowledge; Length; Ligands; Lipids; Lipoprotein Binding; Malignant Neoplasms; Mammals; Mediating; Mediator of activation protein; Molecular; Mus; Mutagenesis; Natural Immunity; Neutrophilia; Operative Surgical Procedures; Pathogenesis; Pattern; Peptides; Peptidoglycan; Peripheral Blood Mononuclear Cell; Pertussis Toxin; Phase; Play; Polymyxin B; Production; Property; Protein C; Protein Isoforms; Publishing; Reporting; Research; Rheumatoid Arthritis; Role; SR-BI receptor; Sepsis Syndrome; Serum amyloid A protein; Signal Transduction; Sterility; Study of serum; TLR4 gene; Therapeutic; Tissues; Toll-Like Receptor 2; Transgenes; Transgenic Mice; Work; activating transcription factor; analog; base; chromatin immunoprecipitation; cytokine; fMet-Leu-Phe receptor; human disease; in vivo; interleukin-23; lipoteichoic acid; macrophage; monocyte; mouse formyl peptide receptor 2; mouse model; mutant; novel; pathogen; peripheral blood; public health relevance; receptor; receptor for advanced glycation endproducts; reconstitution; research study; response