Mechanisms of Homeostasis and Invasive Cell Migration in Skin Tumorigenesis

皮肤肿瘤发生中的稳态和侵袭细胞迁移机制

• 批准号: 8264024
• 负责人: Markus Schober
• 金额: $ 24.9万
• 依托单位: NEW YORK UNIVERSITY SCHOOL OF MEDICINE
• 依托单位国家: 美国
• 财政年份: 2011
• 资助国家: 美国
• 起止时间: 2011-09-01 至 2014-08-31
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/8264024
• 关键词: Anus; Apoptosis; Automobile Driving; Benign; Candidate Disease Gene; Carcinoma; Cell Survival; Cell-Matrix Junction; Cells; Data; Development; Disease; Disseminated Malignant Neoplasm; Epidermis; Epithelial; Epithelium; Equilibrium; Focal Adhesion Kinase 1; Focal Adhesions; Gene Expression; Genes; Genital system; Goals; Growth; Growth Factor; HRAS gene; Homeostasis; Human; Instruction; Integrins; Lead; Link; Literature; Malignant - descriptor; Malignant Conversion; Malignant Neoplasms; Mediating; Metastatic Carcinoma; Metastatic Squamous Cell Carcinoma; Molecular; Molecular Target; Mus; Mutant Strains Mice; Neoplasm Metastasis; Pathway interactions; Play; Probability; Proteomics; Receptor Protein-Tyrosine Kinases; Reporting; Role; Signal Transduction; Skin; Skin Carcinogenesis; Skin Carcinoma; Squamous cell carcinoma; Technology; Testing; Therapeutic; Tissues; Transcription Factor 3; Transducers; Tumor Cell Line; Tumor Markers; Tumor Suppressor Genes; Tyrosine; adhesion receptor; base; carcinogenesis; cell motility; combinatorial; human disease; keratinocyte; migration; neoplastic; novel; novel therapeutic intervention; prevent; programs; receptor; research study; therapeutic target; tumor; tumor initiation; tumor progression; tumorigenesis