Role of Adenosine Receptors in Cardiac Failure and Protection

腺苷受体在心力衰竭和保护中的作用

• 批准号: 8241982
• 负责人: ARTHUR M FELDMAN
• 金额: $ 28.92万
• 依托单位: THOMAS JEFFERSON UNIVERSITY
• 依托单位国家: 美国
• 财政年份: 2011
• 资助国家: 美国
• 起止时间: 2011-04-01 至 2012-01-27
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/8241982
• 关键词: Adenosine; Adenylate Cyclase; Adrenergic Agents; Adrenergic Receptor; Affect; Agonist; Apoptosis; Calcium; Cardiac; Cardiovascular Physiology; Cardiovascular system; Clinical Research; Core Facility; Data; Development; Disease; Epidemic; Extracellular Matrix; Family; Functional disorder; G Protein-Coupled Receptor Signaling; G-Protein-Coupled Receptors; GRK5 gene; GTP-Binding Proteins; Gene Transfer; Goals; Heart; Heart failure; Homeostasis; Hospitalization; Human; Hypertrophy; Image; Individual; Infarction; Inflammatory Response; Injury; Ischemia; Laboratories; Ligands; Mediating; Mediator of activation protein; Modeling; Mus; Myocardial; Myocardial Ischemia; Myocardium; Norepinephrine; Operative Surgical Procedures; Pathway interactions; Patients; Phenotype; Physiological; Physiology; Play; Process; Proteins; Proto-Oncogene Proteins c-akt; Purine Nucleosides; Purinergic P1 Receptors; Reagent; Receptor Activation; Reperfusion Therapy; Role; Safety; Sarcoplasmic Reticulum; Secondary to; Signal Pathway; Signal Transduction; Signaling Molecule; Stress; Technology; Testing; Tissues; Transgenic Mice; Transgenic Model; Tumor Necrosis Factor-alpha; Work; adenosine receptor activation; adrenergic; cellular development; improved; injury and repair; member; mouse model; novel; novel strategies; overexpression; prevent; receptor; regenerative; response; stressor; tool; transgene expression