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Prostate Cancer Parasitism of the HSC "niche" as a Molecular Mechanism for Ost

Prostate Cancer Parasitism of the HSC "niche" as a Molecular Mechanism for Ost
前列腺癌 HSC“生态位”的寄生作为 Ost 的分子机制
批准号：
8284222
负责人：
RUSSELL S TAICHMAN
金额：
$22.01万
依托单位：
UNIVERSITY OF MICHIGAN AT ANN ARBOR
依托单位国家：
美国
项目类别：
财政年份：
2004
资助国家：
美国
项目状态：
未结题
起止时间：
2004-06-05 至
来源：
https://reporter.nih.gov/project-details/8284222
关键词：
ANXA2 geneAccountingAdhesionsAngiogenic SwitchAnimalsAntibodiesAreaBindingBiological AssayBiologyBone MarrowBone Marrow TransplantationCXC ChemokinesCXCR4 ReceptorsCXCR4 geneCellsDataEndothelial CellsEngraftmentEventFoundationsFundingGrowthHematopoietic stem cellsHome environmentHomingHoming BehaviorImplantIndiumInvadedInvestigationLigandsLinkLocationMalignant NeoplasmsMalignant neoplasm of prostateMarrowMetastatic Neoplasm to the BoneMetastatic Prostate CancerModelingMolecularNeoplasm MetastasisOsteoblastsPathway interactionsPeptidesPisum sativumPlayPrimary NeoplasmProstateReceptor SignalingRoleRouteSignal PathwaySignal TransductionSkeletonStromal Cell-Derived Factor 1TestingTherapeutic InterventionTimeTissuesWorkbonecancer cellcell growthfootin vivometastatic processneoplastic cellnovelparasitismreceptorstem cell nichetumor

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Mechanisms That Regulate Dormancy of Disseminated Tumor Cells in the Bone Marrow
Administrative Core
Regulation of the PCa Metastatic Phenotype by the HSC Niche
Prostate Cancer Parasitism of the HSC "niche" as a Molecular Mechanism for Ost
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