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Prostate Cancer Parasitism of the HSC "niche" as a Molecular Mechanism for Ost

Prostate Cancer Parasitism of the HSC "niche" as a Molecular Mechanism for Ost
前列腺癌 HSC“生态位”的寄生作为 Ost 的分子机制
批准号：
8284222
负责人：
RUSSELL S TAICHMAN
金额：
$22.01万
依托单位：
UNIVERSITY OF MICHIGAN AT ANN ARBOR
依托单位国家：
美国
项目类别：
财政年份：
2004
资助国家：
美国
项目状态：
未结题
起止时间：
2004-06-05 至
来源：
https://reporter.nih.gov/project-details/8284222
关键词：
ANXA2 geneAccountingAdhesionsAngiogenic SwitchAnimalsAntibodiesAreaBindingBiological AssayBiologyBone MarrowBone Marrow TransplantationCXC ChemokinesCXCR4 ReceptorsCXCR4 geneCellsDataEndothelial CellsEngraftmentEventFoundationsFundingGrowthHematopoietic stem cellsHome environmentHomingHoming BehaviorImplantIndiumInvadedInvestigationLigandsLinkLocationMalignant NeoplasmsMalignant neoplasm of prostateMarrowMetastatic Neoplasm to the BoneMetastatic Prostate CancerModelingMolecularNeoplasm MetastasisOsteoblastsPathway interactionsPeptidesPisum sativumPlayPrimary NeoplasmProstateReceptor SignalingRoleRouteSignal PathwaySignal TransductionSkeletonStromal Cell-Derived Factor 1TestingTherapeutic InterventionTimeTissuesWorkbonecancer cellcell growthfootin vivometastatic processneoplastic cellnovelparasitismreceptorstem cell nichetumor

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Mechanisms That Regulate Dormancy of Disseminated Tumor Cells in the Bone Marrow
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Prostate Cancer Parasitism of the HSC "niche" as a Molecular Mechanism for Ost
Regulation of the PCa Metastatic Phenotype by the HSC Niche
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