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ADULT NEUROHORMONE MECHANISM RESPONSE TO LARVAL CROWDING

ADULT NEUROHORMONE MECHANISM RESPONSE TO LARVAL CROWDING
成虫神经激素机制对幼虫拥挤的反应
批准号:
2854171
负责人:
JACK W DILLWITH
金额:
$10.4万
依托单位国家:
美国
项目类别:
财政年份:
1996
资助国家:
美国
项目状态:
已结题
起止时间:
1996-09-30 至 2000-09-29

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中文摘要
翻译
描述(改编自申请者的摘要):拥挤有一个 对行为、发育、生长和生殖的不利影响 所有动物,特别是脊椎动物和昆虫的过程。即使所有的一切 其他环境条件最优,拥挤,定义为高 种群密度饲养,通常会在这些 流程。因为这些过程的操作依赖于基础 生理和分子机制,拥挤一定会刺激 这些机制中的应激反应。然而,生理学和 拥挤影响这些过程的分子机制是 不太为人所知。一种检查拥挤对城市影响的方法 神经激素的机制是检查机制的每一个组成部分 分开的。通过这种方式,神经内分泌腺的反应可以是 从目标系统中分离出来。这种方法可以是 利用抗心绞痛的脂肪运动激素机制完成的 因为机构的每个部件都有很好的特性 这种夜蛾表现出强烈的行为和脂肪运动激素 对拥挤的反应。申请者工作假说是 拥挤昆虫的脂肪动员能力的增强是由于其 对脂肪运动激素相关机制成分的影响 随着血淋巴中肽激素水平的升高,反应性/接受性 靶系统对荷尔蒙的影响,或者两者兼而有之。
英文摘要
DESCRIPTION (Adapted from the applicant's abstract): Crowding has a detrimental impact on the behavioral, developmental, growth and reproductive processes of all animals especially vertebrates and insects. Even when all other environmental conditions are optimal, crowding, defined as high population density rearing, usually evokes a stress response in these processes. Since the operation of these precesses relies on underlying physiological and molecular mechanisms, crowding must be stimulating a stress response in these mechanisms. However, the physiological and molecular mechanisms through which crowding influences these processes are not well known. One approach to examining the impact of crowding on a neurohormonal mechanism is to examine each component of the mechanism separately. In this way, the response of the neuroendocrine gland can be separated from that of the target systems. This approach can be accomplished with the adipokinetic hormonal mechanism of Anticarsia gemmatalis because each component of the mechanisms is well characterized and this noctuid moth exhibits a strong behavioral and adipokinetic hormonal response to crowding. The applicants working hypothesis is that the enhanced lipid mobilizing capability of crowded insects is due to its influence on components of the adipokinetic hormonal mechanism associated with the level of the peptide hormone in hemolymph, the response/receptivity of the target systems to the hormone, or both.
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