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ER stress: role in myocyte apoptosis and myocardial remodeling

ER stress: role in myocyte apoptosis and myocardial remodeling
内质网应激：在心肌细胞凋亡和心肌重塑中的作用
批准号：
8195839
负责人：
KRISHNA SINGH
金额：
$0.0万
依托单位：
JAMES H QUILLEN VA MEDICAL CENTER
依托单位国家：
美国
项目类别：
财政年份：
2010
资助国家：
美国
项目状态：
已结题
起止时间：
2010-07-01 至 2014-06-30
来源：
https://reporter.nih.gov/project-details/8195839
关键词：
AddressAdrenergic ReceptorAdultAdverse effectsAffectApoptosisApoptoticBrefeldin ACalciumCalcium SignalingCardiacCardiac MyocytesCellsCessation of lifeChronicDataDiseaseDominant-Negative MutationEndoplasmic ReticulumGRP geneGlycogen Synthase KinasesHeartHeart DiseasesHeart failureHeat shock proteinsHomeostasisHospitalizationIn VitroInfusion proceduresInhibitory G-Protein GiIsoproterenolKnockout MiceLeft Ventricular DysfunctionMitochondriaMolecularMorbidity - disease rateMusMuscle CellsMyocardial InfarctionPathway interactionsPatientsPeptide HydrolasesPlayProcessReceptor SignalingRegulationRoleSarcoplasmic ReticulumSignal PathwaySignal TransductionSiteSmall Interfering RNAStressTestingThapsigarginTranscriptional RegulationTransgenic MiceTunicamycinVentricular RemodelingVeteransbeta-adrenergic receptorcaspase 12endoplasmic reticulum stressimprovedin vivoinhibitor/antagonistinsightmortalitynovel therapeuticsoverexpressionprotein foldingprotein misfoldingsensorstress proteinstressortranscription factortreatment strategy

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  • 批准号：
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