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Molecular Mechanisms of Impaired Angiogenesis in Aging Gastric Mucosa

Molecular Mechanisms of Impaired Angiogenesis in Aging Gastric Mucosa
老化胃粘膜血管生成受损的分子机制
批准号：
8195628
负责人：
ANDRZEJ S TARNAWSKI
金额：
$0.0万
依托单位：
VETERANS HEALTH ADMINISTRATION
依托单位国家：
美国
项目类别：
财政年份：
2010
资助国家：
美国
项目状态：
已结题
起止时间：
2010-04-01 至 2014-03-31
来源：
https://reporter.nih.gov/project-details/8195628
关键词：
5&apos-AMP-activated protein kinaseAcetylationAcuteAge ReportingAge-YearsAgingAlbuminsAmericanAngiogenesis InhibitionAngiogenesis InhibitorsAngiogenic FactorAngiostatinsAntibodiesApoptosisAreaArthritisAssesBindingBiochemicalBlood VesselsBlood capillariesCREB1 geneCell NucleusCell physiologyCellsCharacteristicsChronicCicatrixClinicalComplementary DNAConnective TissueDNA BindingDermalDown-RegulationDuodenal UlcerEconomicsEndostatinsEndothelial CellsEnzymesEpithelialEquilibriumEthanolExhibitsExtravasationFluoresceinFundingGastric mucosaGastric ulcerGastritisGelatinase BGene ActivationGene ExpressionGenesGenetic TranscriptionGoalsGranulation TissueHIF1A geneHealedHealthHeat shock proteinsHeat-Shock Proteins 90HumanHypoxiaHypoxia Inducible FactorImageImageryImpaired wound healingImpairmentImportinsIn VitroIncidenceIndomethacinInfarctionInjuryInvestigationLiteratureMediatingMolecularMorbidity - disease rateMyocardiumNatural regenerationNon-Steroidal Anti-Inflammatory AgentsNuclearNutrientOrganOxygenPainPatientsPeptic UlcerPericytesPermeabilityPhosphorylationPhysiologicalPlasmidsPopulationPredispositionPrevalenceProperdinProteinsPublicationsRattusRelative (related person)ResearchResearch DesignResolutionRoleSTAT3 geneSerumSiteSmall Interfering RNAStimulusStomachStructureTechniquesTechnologyTestingTherapeutic InterventionTimeTissuesTranscriptional ActivationUlcerUp-RegulationVascular Endothelial Growth Factor Receptor-2Vascular Endothelial Growth FactorsVascular PermeabilitiesVeteransWorkWound Healingage relatedalcohol responsealpha Karyopherinsangiogenesisannexin A5autocrinebasecapillarydensitygene therapyhealinghigh riskhypoxia inducible factor 1improvedin vivoindexinginjuredinstrumentmortalitymouse modelmutantneutralizing antibodynovelnovel therapeutic interventionnovel therapeuticsoverexpressionpatient populationpromoterprotein expressionprotein kinase Preceptorresponseresponse to injurystemtranscription factortreatment strategy

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