STATs as Key Targets in Tumor Angiogenesis

STATs 作为肿瘤血管生成的关键靶点

• 批准号: 7789637
• 负责人: ANDREAS FRIEDL
• 金额: --
• 依托单位: WM S. MIDDLETON MEMORIAL VETERANS HOSP
• 依托单位国家: 美国
• 财政年份: 2009
• 资助国家: 美国
• 起止时间: 2009-04-01 至 2013-03-31
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/7789637
• 关键词: Ablation; Activator Appliances; Age; Angiogenesis Inhibition; Angiogenesis Inhibitors; Angiogenic Factor; Animals; Blood Vessels; Brain; Brain Neoplasms; Breeding; Budgets; Bypass; Cancerous; Cardiovascular Diseases; Cell Proliferation; Cell Survival; Cessation of life; Clinical Trials; Computer Retrieval of Information on Scientific Projects Database; Data; Databases; Development; Dimerization; Disease; Dominant-Negative Mutation; Drug Delivery Systems; Drug Design; Endothelial Cells; Event; Extracellular Matrix; FGF2 gene; FGF8 gene; Fibroblast Growth Factor; Fibroblast Growth Factor Receptors; Genetic; Grant; Growth; Health; Healthcare; Heart Diseases; Human; Immunofluorescence Microscopy; In Vitro; Incidence; Intervention; Knock-out; Knowledge; Laboratories; Life; Lung Neoplasms; MAP Kinase Gene; Malignant Neoplasms; Malignant neoplasm of lung; Measures; Mediating; Mediator of activation protein; Pathway interactions; Patients; Pattern; Physiologic Neovascularization; Population; Population Group; Proteins; Recruitment Activity; Regulation; Reliance; Research; Resistance; Resistance development; Role; STAT protein; STAT1 gene; STAT3 gene; STAT5A gene; Sampling; Series; Signal Pathway; Signal Transduction; Stimulation of Cell Proliferation; Stroke; System; Testing; Therapeutic; Therapeutic Intervention; Tissues; Transducers; Tube; Tumor Angiogenesis; Validation; Vascular Endothelial Growth Factors; Veterans; Work; abstracting; angiogenesis; cancer cell; cancer therapy; cancer type; cell motility; cell type; effective therapy; expectation; experience; human disease; improved; in vivo; inhibitor/antagonist; killings; mouse model; mutant; neoplastic cell; novel therapeutic intervention; novel therapeutics; paracrine; pre-clinical; prevent; programs; protein function; public health relevance; response; small molecule; success; therapeutic target; transcription factor; tumor; tumor growth