Characterization of cell GBV-C envelope glycoprotein interactions

细胞 GBV-C 包膜糖蛋白相互作用的表征

• 批准号: 8195612
• 负责人: Jack T. Stapleton
• 金额: --
• 依托单位: IOWA CITY VA MEDICAL CENTER
• 依托单位国家: 美国
• 财政年份: 2009
• 资助国家: 美国
• 起止时间: 2009-04-01 至 2013-03-31
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/8195612
• 关键词: Address; Adverse effects; Anti-Retroviral Agents; Antiviral Agents; Antiviral Therapy; B-Lymphocytes; Binding; CCR5 gene; CD4 Lymphocyte Count; CD4 Positive T Lymphocytes; CD8B1 gene; CXCR4 gene; Cause of Death; Cell Culture Techniques; Cell membrane; Cells; Chemokine (C-C Motif) Receptor 5; Clinical; Clinical Research; Communicable Diseases; Data; Development; Disease; Disease Progression; Down-Regulation; Drug resistance; Effectiveness; Environmental Risk Factor; Epidemic; Family member; Flaviviridae; Functional disorder; GB virus; GB virus C; Genetic Polymorphism; Genetic Transcription; Glycoproteins; HIV; HIV Infections; HIV therapy; HIV vaccine; HIV-1; Health; Healthcare Systems; Hepatic; Hepatitis C virus; Highly Active Antiretroviral Therapy; Homeostasis; Human; Human Virus; IL2RA gene; Immune; Immune response; In Vitro; Incubated; Individual; Infection; Interleukin-2; Lead; Licensing; Lymphocyte; Measures; Mediating; Membrane Proteins; Meta-Analysis; Microbe; Natural History; Nonstructural Protein; Outcome; Patients; Peripheral Blood Mononuclear Cell; Pharmaceutical Preparations; Prevention strategy; Proteins; Provider; Receptor Cell; Regulation; Resistance development; Retroviridae; Risk; Site; Surface; System; T-Cell Activation; T-Cell Receptor; T-Lymphocyte; Toxic effect; United States; Vertical Disease Transmission; Viral; Viral Proteins; Virus; antiretroviral therapy; base; cellular development; chemokine; chemokine receptor; cohort; cost; cytokine; improved; inhibitor/antagonist; insight; microbial; new therapeutic target; novel strategies; novel therapeutic intervention; particle; public health relevance; receptor; receptor expression; release factor; response; vaccine development